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Kiminori Sugino

Researcher at Yokohama City University

Publications -  175
Citations -  4319

Kiminori Sugino is an academic researcher from Yokohama City University. The author has contributed to research in topics: Thyroid & Thyroid carcinoma. The author has an hindex of 28, co-authored 151 publications receiving 3674 citations. Previous affiliations of Kiminori Sugino include Nippon Medical School & Yokohama City University Medical Center.

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Lymph Node Metastasis From 259 Papillary Thyroid Microcarcinomas: Frequency, Pattern of Occurrence and Recurrence, and Optimal Strategy for Neck Dissection

TL;DR: Patients who have PTMC presenting with palpable lymphadenopathy should have therapeutic node dissection, and patients without palpable nodes underwent prophylactic node dissections, to establish the optimal strategy for neck dissection in these patients.
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Thyroid Lobectomy for Papillary Thyroid Cancer: Long-term Follow-up Study of 1,088 Cases

TL;DR: It is concluded that lobectomy is a valid alternative to total thyroidectomy for the treatment of PTC patients who are younger than aged 45 years, whose tumor diameter is 40 mm or less, and who do not have clinical lymph node metastasis or extrathyroidal invasion.
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Immediate causes of death in thyroid carcinoma: clinicopathological analysis of 161 fatal cases.

TL;DR: Based on this knowledge, several palliative procedures may be worth considering to improve survival and quality of life in patients with advanced thyroid carcinoma.
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Prognostic Factors and Treatment Outcomes for Anaplastic Thyroid Carcinoma: ATC Research Consortium of Japan Cohort Study of 677 Patients

TL;DR: Long-term survival is possible for selected patients with ATC and to determine the treatment strategy, UICC stage (disease extent) and other prognostic factors (e.g., biologic malignancy grade) should be considered.
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Low Frequency of BRAFT1796A Mutations in Childhood Thyroid Carcinomas

TL;DR: Analysis of PTCs in 31 Japanese and 48 post-Chernobyl Ukrainian thyroid carcinomas could detect no positive association between BRAF(T1796A) mutations and clinical parameters in the childhood carcinomas, suggesting that a low prevalence of BRAFs is a common feature of P TCs in children regardless of radiation exposure levels.