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Kirk W. Barron
Researcher at University of Oklahoma Health Sciences Center
Publications - 46
Citations - 1897
Kirk W. Barron is an academic researcher from University of Oklahoma Health Sciences Center. The author has contributed to research in topics: Mean arterial pressure & Vasodilation. The author has an hindex of 25, co-authored 46 publications receiving 1825 citations. Previous affiliations of Kirk W. Barron include University of Kentucky & University of Iowa.
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Corticosterone delivery to the amygdala increases corticotropin-releasing factor mRNA in the central amygdaloid nucleus and anxiety-like behavior
TL;DR: Findings support the involvement of the amygdala in anxiety-like behaviors in response to chronically elevated corticosterone and suggests that elevated glucocorticoids may increase anxiety by inducing CRF expression in the CeA.
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Modulation of intrinsic cardiac neurons by spinal cord stimulation: implications for its therapeutic use in angina pectoris.
Robert D. Foreman,Bengt Linderoth,Jeffrey L. Ardell,Kirk W. Barron,Margaret J. Chandler,Stephen S. Hull,Gert J. Terhorst,Mike J. L. DeJongste,John A Armour +8 more
TL;DR: Modulation of the intrinsic cardiac nervous system might contribute to the therapeutic effects of SCS in patients with angina pectoris, as it modifies the capacity of intrinsic cardiac neurons to generate activity.
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Stereotaxic localization of corticosterone to the amygdala enhances hypothalamo-pituitary-adrenal responses to behavioral stress.
TL;DR: In conclusion, direct administration of corticosterone to the amygdala increases plasma Corticosterone in response to a behavioral stressor without altering the diurnal rhythm in plasma cortic testosterone.
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Cutaneous vasodilation during dorsal column stimulation is mediated by dorsal roots and CGRP
TL;DR: It is proposed that DCS antidromically activates afferent fibers in the dorsal roots, thus causing peripheral release of CGRP, which produces cutaneous vasodilation.
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Vocal indices of stress: a review.
TL;DR: Stress types, gender, and individual differences in baseline autonomic tone may explain the primarily equivocal findings of effects of stressor exposure or perceived stress on voice; and as such, the article concludes with a discussion of directions for future study.