K
Konrad J. Falke
Researcher at Free University of Berlin
Publications - 60
Citations - 5941
Konrad J. Falke is an academic researcher from Free University of Berlin. The author has contributed to research in topics: ARDS & Respiratory distress. The author has an hindex of 28, co-authored 60 publications receiving 5769 citations.
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Journal ArticleDOI
Inhaled Nitric Oxide for the Adult Respiratory Distress Syndrome
TL;DR: Inhalation of nitric oxide by patients with severe adult respiratory distress syndrome reduces the pulmonary-artery pressure and increases arterial oxygenation by improving the matching of ventilation with perfusion, without producing systemic vasodilation.
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Immunologic and hemodynamic effects of "low-dose" hydrocortisone in septic shock: a double-blind, randomized, placebo-controlled, crossover study.
Didier Keh,Thomas Boehnke,Steffen Weber-Cartens,Christina Schulz,Olaf Ahlers,Sven Bercker,Hans-Dieter Volk,Wolf-Dietrich Doecke,Konrad J. Falke,Herwig Gerlach +9 more
TL;DR: Hydrocortisone therapy restored hemodynamic stability and differentially modulated the immunologic response to stress in a way of antiinflammation rather than immunosuppression.
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Time-course and dose-response of nitric oxide inhalation for systemic oxygenation and pulmonary hypertension in patients with adult respiratory distress syndrome
TL;DR: Inhalation of nitric oxide for improvement of oxygenation in severe ARDS should be performed using lower doses, with lower risk of toxic side effects.
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Influence of Positioning on Ventilation-Perfusion Relationships in Severe Adult Respiratory Distress Syndrome
TL;DR: It is concluded that improvement of oxygenation during pressure-controlled mechanical ventilation in the prone position is due to a shift of blood flow away from shunt regions, thus increasing areas with normal VA/Q.
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Autoinhalation of nitric oxide after endogenous synthesis in nasopharynx
TL;DR: In volunteers, 50-70% of the NO was resorbed by the lungs; ventilated patients were deprived of NO autoinhalation, suggesting bacteria in the nose may take part in endogenous NO synthesis.