L
L M Buja
Researcher at University of Texas Health Science Center at Houston
Publications - 321
Citations - 13641
L M Buja is an academic researcher from University of Texas Health Science Center at Houston. The author has contributed to research in topics: Myocardial infarction & Myocyte. The author has an hindex of 63, co-authored 304 publications receiving 13331 citations. Previous affiliations of L M Buja include Baylor College of Medicine & University of Texas MD Anderson Cancer Center.
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Specific platelet mediators and unstable coronary artery lesions. Experimental evidence and potential clinical implications.
TL;DR: It is believed that unstable angina and acute myocardial infarction are a continuum in relation to the process of coronary artery thrombosis and vasoconstriction.
Journal Article
Apoptosis and necrosis. Basic types and mechanisms of cell death.
TL;DR: These insights point the way for further research to establish definitive causes of specific types of cell injury and cell death, and they provide important clues for the design of improved diagnostic approaches and therapeutic interventions.
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Abnormal I-123 metaiodobenzylguanidine myocardial washout and distribution may reflect myocardial adrenergic derangement in patients with congestive cardiomyopathy.
E Henderson,Joel K. Kahn,James R. Corbett,D. E. Jansen,John J. Pippin,Padmakar V. Kulkarni,Valentina Ugolini,Marvin S. Akers,Christopher L. Hansen,L M Buja,Robert W. Parkey,James T. Willerson +11 more
TL;DR: It is concluded that the myocardial distribution and kinetics of MIBG in images obtained from patients with CM differ significantly from those of controls and that the M IBG patterns may be used as a relatively noninvasive means to evaluate the severity of altered adrenergic innervation in the hearts of these patients.
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The emerging spectrum of cardiopulmonary pathology of the coronavirus disease 2019 (COVID-19): Report of 3 autopsies from Houston, Texas, and review of autopsy findings from other United States cities.
L M Buja,Dwayne A. Wolf,Bihong Zhao,Bindu Akkanti,Michelle McDonald,Laura Lelenwa,Noah Reilly,Giulia Ottaviani,M. Tarek Elghetany,Daniel Ocazionez Trujillo,Gabriel M Aisenberg,Mohammad Madjid,Biswajit Kar +12 more
TL;DR: The autopsy findings support the concept that the pathogenesis of severe COVID-19 disease involves direct viral-induced injury of multiple organs, including heart and lungs, coupled with the consequences of a procoagulant state with coagulopathy.
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Accumulation of Unesterified Arachidonic Acid in Ischemic Canine Myocardium Relationship to a Phosphatidylcholine Deacylation-Reacylation Cycle and the Depletion of Membrane Phospholipids
TL;DR: It is postulated that the defective reacylation of arachidonate into phosphatidylcholine may contribute to the net loss of membrane phospholipid during myocardial ischemia, and suggests that the accumulation of archidonate may be a more sensitive measure of phospholIPid degradation than the decrease in total phospholipopid content in ischemic canine myocardium.