L
Lei Li
Researcher at Nanjing Medical University
Publications - 73
Citations - 1431
Lei Li is an academic researcher from Nanjing Medical University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 16, co-authored 52 publications receiving 742 citations.
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Journal ArticleDOI
A purified membrane protein from Akkermansia muciniphila or the pasteurised bacterium blunts colitis associated tumourigenesis by modulation of CD8+ T cells in mice
Lijuan Wang,Lei Tang,Yiming Feng,Suying Zhao,Mei Han,Chuan Zhang,Ge-hui Yuan,Jun Zhu,Shuyuan Cao,Qian Wu,Lei Li,Zhan Zhang +11 more
TL;DR: Data indicate that pasteurised A. muciniphila or Amuc_1100 can blunt colitis and CAC through the modulation of CTLs.
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Caffeic acid ameliorates colitis in association with increased Akkermansia population in the gut microbiota of mice
TL;DR: Dietary CaA could ameliorate colonic pathology and inflammation in DSS colitis mice, and it might be associated with a proportional increase in Akkermansia.
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Chlorogenic Acid Ameliorates Experimental Colitis by Promoting Growth of Akkermansia in Mice
TL;DR: Chlorogenic acid acted as the intestine-modifying gut microbial community structure, resulting in a lower intestinal and systemic inflammation and also improving the course of the DSS-induced colitis, which is associated with a proportional increase in Akkermansia.
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Photocatalytic degradation of microcystin-LR by modified TiO2 photocatalysis: A review.
TL;DR: Progress has been summarized in the development of heterogeneousTiO2-based photocatalysts for MC-LR photodegradation under visible, UV, or solar light and some main modification methods for improving the drawbacks and performance of TiO2 nanoparticle were highlighted.
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P38/NF-κB/Snail Pathway Is Involved in Caffeic Acid-Induced Inhibition of Cancer Stem Cells-Like Properties and Migratory Capacity in Malignant Human Keratinocyte
TL;DR: CaA attenuates the migratory capability and CSCs-like Properties of malignant human keratinocyte, in which, p38-mediated down-regulation of NF-κB/snail signal pathway is involved.