L
Lewis S. Blevins
Researcher at University of California, San Francisco
Publications - 94
Citations - 2590
Lewis S. Blevins is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Pituitary adenoma & Adenoma. The author has an hindex of 25, co-authored 90 publications receiving 2273 citations. Previous affiliations of Lewis S. Blevins include Vanderbilt University Medical Center & Vanderbilt University.
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Effect of Minimally Invasive Radioguided Parathyroidectomy on Efficacy, Length of Stay, and Costs in the Management of Primary Hyperparathyroidism
TL;DR: The MIRP technique resulted in excellent cure rates for primary hyperparathyroidism while simultaneously decreasing operative time and hospital stays and resulted in significant cost reductions without compromising patient safety.
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Synergistic effect of adrenal steroids and angiotensin II on plasminogen activator inhibitor-1 production.
Nancy J. Brown,Kyungsoo Kim,Yan Qun Chen,Lewis S. Blevins,John H. Nadeau,Steven G. Meranze,Douglas E. Vaughan +6 more
TL;DR: The hypothesis that aldosterone modulates the effect of Ang II on PAI-1 expression in vitro and in vivo in humans is supported.
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The Low Dose (1-μg) Adrenocorticotropin Stimulation Test in the Evaluation of Patients with Suspected Central Adrenal Insufficiency
TL;DR: It is suggested that the 1- microg ACTH stimulation test replace the conventional 250-microg test when evaluating for central adrenal insufficiency and pharmaceutical companies should be encouraged to provide synthetic ACTH in 1- Microg vials.
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Outcomes of Therapy for Cushing’s Disease due to Adrenocorticotropin-Secreting Pituitary Macroadenomas
TL;DR: The results indicate that ACTH-secreting macroadenomas are more refractory to conventional treatments than are microadenoma patients, and pharmacological agents and adrenalectomy were effective in the management of hypercortisolism in patients with residual and recurrent disease.
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The effects of mild ethanol intoxication on the hypothalamic-pituitary-adrenal axis in nonalcoholic men.
TL;DR: In summary, mildly intoxicating doses of ethanol did not stimulate the HPA axis in six of eight subjects, however, mild intoxication significantly impaired oCRH-stimulated ACTH/cortisol secretion and it is speculated that mild intoxication with ethanol may impair the ability of the H PA axis to respond to physiological stressors.