L
Limei Hu
Researcher at Chongqing University
Publications - 6
Citations - 70
Limei Hu is an academic researcher from Chongqing University. The author has contributed to research in topics: Computer science & Wireless network. The author has an hindex of 2, co-authored 3 publications receiving 14 citations.
Papers
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Journal ArticleDOI
Status Update in IoT Networks: Age-of-Information Violation Probability and Optimal Update Rate
TL;DR: This work focuses on characterizing the violation probability of peak AoI and AoI in IoT systems, where a sensor delivers updates to a monitor under M/M/1 and M/D/1 queues with first-come–first-served policy.
Journal ArticleDOI
A Robust Diffusion Estimation Algorithm for Asynchronous Networks in IoT
TL;DR: A distributed diffusion kernel risk-sensitive loss (d-KRSL) algorithm is proposed, which can achieve a good performance in asynchronous networks with varying topology, and maintains the robustness to both Gaussian and impulsive noise.
Proceedings ArticleDOI
Optimal Status Update in IoT Systems: An Age of Information Violation Probability Perspective
TL;DR: In this article, the AoI violation probability in the M/M/1 queue with first-come-first-served (FCFS) policy was investigated in real-time IoT networks.
Journal ArticleDOI
Analysis of Age of Information in Dual Updating Systems
Zhengchuan Chen,Dapeng Deng,Howard H. Yang,Nikolaos Pappas,Limei Hu,Yunjian Jia,Min Wang,Tony Q. S. Quek +7 more
TL;DR: Numerical results show that the two dual-queue systems also outperform the M/M/2 single queue dual-server system with optimized arrival rate in terms of average AoI and PAoI.
Journal ArticleDOI
Short-term exposure of HFD depresses intestinal cholinergic anti-inflammatory activity through hypothalamic inflammation in mice.
TL;DR: In this paper , the effect of high-fat diet exposure on intestinal cholinergic anti-inflammatory activity in mice was evaluated by heart rate variability, and the results revealed that HFD challenge depresses intestinal choline antiinflammatory activity, which is mediated by hypothalamic inflammation.