Lívia Corrêa Barroso

TL;DR: Activation of Mas receptors decreases neutrophil influx and cytokine production and causes significant amelioration of arthritis in experimental models ofthritis in rats and mice.

TL;DR: It is shown that ARBs have potent anti-inflammatory effects in animal models of arthritis and are accompanied by functional improvement of the joint, as seen by reduced joint hypernociception.

TL;DR: Results demonstrate, for the first time, that angiotensin-(1–7) activates events that are crucial for resolution of the inflammatory process of asthma and promotion of the return of lung homeostasis, indicating Ang-(1-7) as novel endogenous inflammation-resolving mediator.

TL;DR: It is suggested that exogenous activation of the Mas receptor protects from ADR-induced nephropathy and contributes to the beneficial effects of AT1 receptor blockade.

TL;DR: Administration of AVE0991 promoted renoprotective effects, as seen by improvement of function, decreased tissue injury, prevention of local and remote leucocyte infiltration, and release of the chemokine, CXCL1.