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Luis M. Montaño

Researcher at National Autonomous University of Mexico

Publications -  104
Citations -  1450

Luis M. Montaño is an academic researcher from National Autonomous University of Mexico. The author has contributed to research in topics: Receptor & Myocyte. The author has an hindex of 20, co-authored 97 publications receiving 1245 citations. Previous affiliations of Luis M. Montaño include McMaster University & CINVESTAV.

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Relaxation of Androgens on Rat Thoracic Aorta: Testosterone Concentration Dependent Agonist/Antagonist l-Type Ca2+ Channel Activity, and 5β-Dihydrotestosterone Restricted to l-Type Ca2+ Channel Blockade

TL;DR: The data show that 5beta-DHT-induced vasorelaxation is due to its selective blockade on L-VOCCs (from nm to microm concentrations), but testosterone- induced vasore laxation involves concentration-dependent additional mechanisms: acting as an L- VOCCs antagonist at low concentrations, and increasing [Ca(2+)]i and cAMP production at high concentrations.
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A non-invasive method to isolate the neuronal linage from the nasal epithelium from schizophrenic and bipolar diseases

TL;DR: Data support that neural precursors obtained from a nasal exfoliate are an excellent experimental model to later approach studies on biomarkers, neural development and cellular alterations in the pathophysiology of SZ and BD.
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Inhibitory interactions between 5-HT3 and P2X channels in submucosal neurons

TL;DR: Kinetic and pharmacological properties of I(5-HT+ATP) indicate that they are carried through 5-HT(3) and P2X channels, in agreement with a model that considers that 5- HT(3), P2x channels are in functional clusters and that these channels might directly inhibit each other.
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Androgens Induce Relaxation of Contractile Activity in Pregnant Human Myometrium at Term: A Nongenomic Action on L-Type Calcium Channels

TL;DR: Observations demonstrate that androgens may play a crucial role in maintaining pregnancy and the blockade of l-type calcium channels seems to be involved in the nongenomic relaxing action of androgens.
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Androgens are bronchoactive drugs that act by relaxing airway smooth muscle and preventing bronchospasm.

TL;DR: It is suggested that androgen-induced brochorelaxation was mediated via decreased Ca(2+) influx through L-type Ca( 2+)channels but additional Ca(1+) entry blockade may be involved.