L
Lung Kun Yeh
Researcher at Memorial Hospital of South Bend
Publications - 10
Citations - 660
Lung Kun Yeh is an academic researcher from Memorial Hospital of South Bend. The author has contributed to research in topics: Keratocan & Transplantation. The author has an hindex of 7, co-authored 10 publications receiving 602 citations. Previous affiliations of Lung Kun Yeh include Chang Gung University & Bascom Palmer Eye Institute.
Papers
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Journal ArticleDOI
How Does Amniotic Membrane Work
Scheffer C.G. Tseng,Edgar M. Espana,Tetsuya Kawakita,Mario A. Di Pascuale,Wei Li,Hua He,Tzong Shyne Liu,Tae Hee Cho,Ying Ying Gao,Lung Kun Yeh,Chia-Yang Liu +10 more
TL;DR: This review describes how the actions inherently operate in utero and how amniotic membrane transplantation aims to recreate such a fetal environment to exert these actions by insulating the surgical site from the host environment.
Journal Article
Intraoperative Mitomycin C and Amniotic Membrane Transplantation for Fornix Reconstruction
Scheffer C.G. Tseng,M.A. Di Pascuale,Edgar M. Espana,Tetsuya Kawakita,T. Liu,H.–T. Cho,Lung Kun Yeh +6 more
TL;DR: Intraoperative application of mitomycin C is an effective means to reduce chronic and deep-seated conjunctival inflammation, and helps amniotic membrane restore a deep fornix after symblepharon lysis, even in eyes that had a failed MMG.
Journal ArticleDOI
Soluble lumican glycoprotein purified from human amniotic membrane promotes corneal epithelial wound healing.
Lung Kun Yeh,Wei-Li Chen,Wei-Li Chen,Wei Li,Edgar M. Espana,J. Ouyang,Tetsuya Kawakita,Winston W.-Y. Kao,Scheffer C.G. Tseng,Chia-Yang Liu +9 more
TL;DR: The diversified functions of lumican include modulation of epithelial cells in wound healing and serving as an extracellular matrix component in human amniotic membrane.
Journal ArticleDOI
Calcium-induced abnormal epidermal-like differentiation in cultures of mouse corneal-limbal epithelial cells.
TL;DR: Mouse corneal-limbal epithelial sheets can be used for initiating primary cultures, and their differentiation is promoted, whereas growth is suppressed, by a high Ca(2+) concentration, even during enzymatic digestion.
Journal ArticleDOI
Pax6 overexpression suppresses cell proliferation and retards the cell cycle in corneal epithelial cells.
J. Ouyang,Ying Cheng Shen,Lung Kun Yeh,Wei Li,Brad M. Coyle,Chia-Yang Liu,M. Elizabeth Fini,M. Elizabeth Fini +7 more
TL;DR: Inhibition of cell proliferation in Pax6-overexpressing corneal epithelial cell lines and primary cell culture is consistent with the notion that Pax6 plays a role in controlling cornealsomehelial cell dynamics in vivo.