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Luxin Wei

Researcher at Chinese Center for Disease Control and Prevention

Publications -  20
Citations -  567

Luxin Wei is an academic researcher from Chinese Center for Disease Control and Prevention. The author has contributed to research in topics: Cancer & Cumulative dose. The author has an hindex of 12, co-authored 20 publications receiving 535 citations.

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Cancer Mortality in the High Background Radiation Areas of Yangjiang, China during the Period between 1979 and 1995

TL;DR: The mortality of all cancers in HBRA was generally lower than that in the control area, but not statistically significant, and neither homogeneity tests nor trend tests revealed any statistically significant relationship between cancer risk and radiation dose.
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Cancer and non-cancer mortality among Inhabitants in the high background radiation area of Yangjiang, China (1979-1998).

TL;DR: The cumulative HBR dose was not related to the mortality due to cancer or all non-cancer diseases among residents in Yangjiang HBR areas, and liver-cancer mortality was inverselyrelated to the cumulative dose.
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An introductory overview of the epidemiological study on the population at the high background radiation areas in Yangjiang, China.

TL;DR: A historical overview of the studies before the cooperation and the first two phases of the cooperative project involving both Chinese and Japanese scientists is presented.
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A case-control study of nasopharyngeal carcinoma in the high background radiation areas of Yangjiang, China.

TL;DR: Univariate conditional logistic regression analysis showed that NPC risk was associated with the consumption of salted fish, homemade pickles, and fermented soy beans, education levels, the history of chronic rhinitis, and the family history of NPC.
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Dose-effect Relationship of Dicentric and Ring Chromosomes in Lymphocytes of Individuals Living in the High Background Radiation Areas in China

TL;DR: The frequency of Dic + Rc linearly increases over lifetime due to chronic low dose exposure and it is likely that the activation of repair enzymes is not triggered in the present HBRA, and Threshold dose of the induction of chromosome aberrations, if any, is below the present dose level.