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Magdalena Pezzoni

Researcher at National Scientific and Technical Research Council

Publications -  22
Citations -  227

Magdalena Pezzoni is an academic researcher from National Scientific and Technical Research Council. The author has contributed to research in topics: Biofilm & Pseudomonas aeruginosa. The author has an hindex of 9, co-authored 19 publications receiving 137 citations.

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Protective role of extracellular catalase (KatA) against UVA radiation in Pseudomonas aeruginosa biofilms.

TL;DR: In this article, the protection role of KatA catalase in both planktonic cells and biofilms of Pseudomonas aeruginosa against UVA radiation was determined by using the wild-type (PAO1) and an isogenic Catalase deficient strain (katA).
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Optically transparent silver-loaded mesoporous thin film coating with long-lasting antibacterial activity

TL;DR: In this paper, titania-silica bilayer mesoporous thin film (MTF) coatings have been produced and loaded with silver species, and their bactericidal efficiency against Pseudomonas aeruginosa and Staphylococcus aureus was assessed over time.
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Exposure to low doses of UVA increases biofilm formation in Pseudomonas aeruginosa.

TL;DR: It is demonstrated that exposure of planktonic cells to sublethal doses of UVA can increase cell surface hydrophobicity and swimming motility, two parameters known to favor cell adhesion, suggesting that UVA radiation acts, at least in part, by promoting the first stages of biofilm development.
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New envelope stress factors involved in σE activation and conditional lethality of rpoE mutations in Salmonella enterica.

TL;DR: It is demonstrated that UVA radiation induces σE activity in a pathway that is dependent on the stringent response regulator ppGpp, and rpoE has a role in the defence against lethal UVA doses that is mediated by functions that are dependent on and independent of the alternative sigma factor RpoS.
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Exposure to low UVA doses increases KatA and KatB catalase activities, and confers cross-protection against subsequent oxidative injuries in Pseudomonas aeruginosa.

TL;DR: It is demonstrated that exposure to low UVA produced a protective effect against subsequent lethal doses of UVA, sodium hypochlorite and H2O2, demonstrating that different mechanisms are involved in the defence against these oxidative agents, although both genes can beinvolved in the global cellular response.