scispace - formally typeset
M

Manuel Martínez-Luque

Researcher at University of Córdoba (Spain)

Publications -  22
Citations -  1207

Manuel Martínez-Luque is an academic researcher from University of Córdoba (Spain). The author has contributed to research in topics: Rhodobacter & Nitrate reductase. The author has an hindex of 15, co-authored 22 publications receiving 1098 citations.

Papers
More filters
Journal ArticleDOI

Prokaryotic Nitrate Reduction: Molecular Properties and Functional Distinction among Bacterial Nitrate Reductases

TL;DR: Nitrogen is a basic element for life because it is a component of the two preeminent biological macromolecules: proteins and nucleic acids.
Journal ArticleDOI

Bacterial degradation of cyanide and its metal complexes under alkaline conditions.

TL;DR: Cyanase activity was induced during growth with cyanide or cyanate, but not with ammonium or nitrate as the nitrogen source, suggesting that cyanate could be an intermediate in the cyanide degradation pathway, but alternative routes cannot be excluded.
Journal ArticleDOI

Alkaline cyanide degradation by Pseudomonas pseudoalcaligenes CECT5344 in a batch reactor. Influence of pH.

TL;DR: Water containing cyanide was biologically detoxified with the bacterial strain Pseudomonas pseudoalcaligenes CECT5344 in a batch reactor, which constitutes an effective alternative to available physico-chemical methods for the purification of wastewater containing cyanides or cyano-metal complexes.
Journal ArticleDOI

Cyanide metabolism of Pseudomonas pseudoalcaligenes CECT5344: role of siderophores

TL;DR: A proteomic approach was used for the isolation and identification, in this strain, of a protein that was induced in the presence of cyanide, namely CN0, that is involved in siderophore biosynthesis in response to cyanide.
Journal ArticleDOI

Bacterial cyanide degradation is under review: Pseudomonas pseudoalcaligenes CECT5344, a case of an alkaliphilic cyanotroph.

TL;DR: This review provides an overview of how cells deal in the presence of the poison cyanide that irreversible binds to metals causing, among other things, iron-deprivation conditions outside the cell and metalloenzymes inhibition inside the cell.