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Manuel Nieto-Díaz

Researcher at Spanish National Research Council

Publications -  32
Citations -  801

Manuel Nieto-Díaz is an academic researcher from Spanish National Research Council. The author has contributed to research in topics: microRNA & Spinal cord injury. The author has an hindex of 15, co-authored 26 publications receiving 683 citations.

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MicroRNA Dysregulation in the Spinal Cord following Traumatic Injury

TL;DR: It is demonstrated that moderate spinal cord injury induces an extended microRNA downregulation paralleled by an increase in mRNA expression that affects key processes in the pathophysiology of this injury.
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Regional and environmental effects on the species richness of mammal assemblages

TL;DR: In this paper, the authors investigated the relationship between species richness and climate and found that the importance of environmental variables varies with scale: climatic gradients are more important at coarse grain (larger sites), possibly as a result of their effects on species ranges, whereas habitat type is more important in the smaller sites, where the significance of ecological interactions increases.
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MicroRNA dysregulation in spinal cord injury: causes, consequences and therapeutics.

TL;DR: The present article reviews the actual knowledge on how injury affects microRNA expression and the meaning of these changes in the SCI pathophysiology, to finally explore the clinical potential of microRNAs in theSCI.
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Postnatal changes in the growth dynamics of the human face revealed from bone modelling patterns.

TL;DR: This study establishes for the first time the bone modelling pattern of the face and the mandible from adult humans and hypothesizes that these ontogenetic changes would respond to the physiological and physical requirements to enlarge the oral and nasal cavities once maturation of the brain and the closure of the cranial sutures have taken place during craniofacial development.
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Inhibitors of Glioma Growth that Reveal the Tumour to the Immune System.

TL;DR: The ability of gliomas to evade and suppress the host immune system, exhibited at the levels of antigen recognition and immune activation, limiting the effective signaling between glioma and host immune cells is examined.