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Marcelo H Ratto

Researcher at Austral University of Chile

Publications -  79
Citations -  1413

Marcelo H Ratto is an academic researcher from Austral University of Chile. The author has contributed to research in topics: Ovulation & Corpus luteum. The author has an hindex of 20, co-authored 70 publications receiving 1182 citations. Previous affiliations of Marcelo H Ratto include Ross University School of Veterinary Medicine & Temuco Catholic University.

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The nerve of ovulation-inducing factor in semen

TL;DR: It is concluded that OIF in seminal plasma is β-NGF and that it is highly conserved and elucidates a previously unknown pathway for the direct influence of the male on the hypothalamo–pituitary–gonadal axis of the inseminated female.
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Ovulation-inducing factor in seminal plasma: A review

TL;DR: Ovulation-inducing factor in seminal plasma is a potent stimulant of LH secretion, ovulation and luteal gland development, and acts via a systemic rather than a local route.
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Biochemical isolation and purification of ovulation-inducing factor (OIF) in seminal plasma of llamas.

TL;DR: Ovulation-inducing factor was isolated from llama seminal plasma as a 14 kDa protein molecule that elicits a preovulatory LH surge followed by ovulation and CL formation in llamas, suggesting an endocrine effect at the level of the hypothalamus (release of GnRH) or the pituitary (gonadotrophs).
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Ovulation-inducing factor (OIF/NGF) in seminal plasma: a review and update

TL;DR: An update on the progress in the understanding of the nature of OIF/NGF in seminal plasma and its effects on reproductive function in the female is provided, including the effects of dose and route of administration, evidence for ovarian effects in other species, tissue sources and early findings related to the mechanism of action.
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Cetrorelix suppresses the preovulatory LH surge and ovulation induced by ovulation-inducing factor (OIF) present in llama seminal plasma

TL;DR: Cetrorelix (GnRH antagonist) inhibited the preovulatory LH surge induced by OIF in llamas suggesting that LH secretion is modulated by a direct or indirect effect of OIF on GnRH neurons in the hypothalamus.