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Maristela O. Poletini

Researcher at Universidade Federal de Minas Gerais

Publications -  37
Citations -  658

Maristela O. Poletini is an academic researcher from Universidade Federal de Minas Gerais. The author has contributed to research in topics: CLOCK & Circadian rhythm. The author has an hindex of 15, co-authored 34 publications receiving 568 citations. Previous affiliations of Maristela O. Poletini include Florida State University & University of São Paulo.

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Environmental control of biological rhythms: effects on development, fertility and metabolism.

TL;DR: Pernicious alterations of the endogenous temporal order by environmental factors can disrupt the homeostatic function of the circadian timing system, leading to pathophysiology and/or disease.
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Changes in α-estradiol receptor and progesterone receptor expression in the locus coeruleus and preoptic area throughout the rat estrous cycle

TL;DR: Data show that LC neurons express ER and PR and seem to be more sensitive to variations in estradiol than POA, which might be related to the ability ofEstradiol and progesterone in regulating the activity of LC neurons, which could be associated to the control mechanisms of LH and prolactin release.
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Release of norepinephrine in the preoptic area activates anteroventral periventricular nucleus neurons and stimulates the surge of luteinizing hormone.

TL;DR: Estradiol and progesterone activate A2 and LC neurons, respectively, and this is associated with the increased release of NE in the POA and the magnitude of the LH surge, which stimulates LH secretion, at least in part, through activation of anteroventral periventricular neurons.
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Anatomical and functional characterization of clock gene expression in neuroendocrine dopaminergic neurons.

TL;DR: The data suggest that clock gene expression, particularly PER1 and PER2, within NDNs may act to modulate diurnal rhythms of DA release from NDNs in the OVX rat.
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Oxytocin action at the lactotroph is required for prolactin surges in cervically stimulated ovariectomized rats.

TL;DR: The cervical stimulation-induced prolactin surges are maintained even in the absence of oxytocin actions at the lactotroph, which strongly suggests the maintenance of prolact in surges are not dependent upon oxytoc in action at the pituitary gland.