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Markku S. Nieminen

Researcher at University of Helsinki

Publications -  493
Citations -  73499

Markku S. Nieminen is an academic researcher from University of Helsinki. The author has contributed to research in topics: Left ventricular hypertrophy & Heart failure. The author has an hindex of 101, co-authored 492 publications receiving 69361 citations. Previous affiliations of Markku S. Nieminen include Helsinki University Central Hospital & Oulu University Hospital.

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The correlation between symptomatic CMV infection and CMV antigenemia in heart allograft recipients.

TL;DR: High antigenemia levels (>100/50,000) correlate with clinical manifestations of CMV infection, and Quantitative monitoring ofCMV antigenemia may, thus, be helpful in the clinical diagnosis of CMVs infection in heart transplant patients.
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Effects of acute alcohol ingestion on heart rate variability in patients with documented coronary artery disease and stable angina pectoris

TL;DR: In the frequency domain analysis, the high-, low-, and very low frequency components were significantly decreased after alcohol: these changes last longer than the elimination of alcohol.
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Novel mutations in the lamin A/C gene in heart transplant recipients with end stage dilated cardiomyopathy.

TL;DR: This study investigates a homogeneous group of consecutive Finnish heart transplant recipients with end stage DCM and searches for mutations in the lamin A/C gene, which is known to be important aetiological factors in familial DCM.
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Regression of electrocardiographic left ventricular hypertrophy predicts regression of echocardiographic left ventricular mass: the LIFE study.

TL;DR: Patients with no significant decrease and patients with regression of ECG LVH had stepwise greater absolute decreases in LV mass and were more likely to decrease LV mass, even after adjusting for possible effects of baseline and change in systolic and diastolic pressures.
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Lipopolysaccharide, a possible molecular mediator between periodontitis and coronary artery disease

TL;DR: In particular in periodontitis patients, subgingival microbial burden contributes to endotoxemia, suggesting LPS is a possible molecular mediator betweenperiodontitis and CAD.