M
Marta Otero-Viñas
Researcher at University of Vic
Publications - 22
Citations - 1492
Marta Otero-Viñas is an academic researcher from University of Vic. The author has contributed to research in topics: Vascular smooth muscle & Wound healing. The author has an hindex of 11, co-authored 20 publications receiving 1072 citations. Previous affiliations of Marta Otero-Viñas include Boston University & University of Gothenburg.
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Journal ArticleDOI
Research Techniques Made Simple: Analysis of Collective Cell Migration Using the Wound Healing Assay
Ayman Grada,Marta Otero-Viñas,Marta Otero-Viñas,Francisco Prieto-Castrillo,Zaidal Obagi,Vincent Falanga +5 more
TL;DR: The wound healing assay, also known as the "in vitro scratch assay" is explained as a simple, versatile, and cost-effective method to study collective cell migration and wound healing.
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Transforming growth factor beta (TGF-β) isoforms in wound healing and fibrosis.
TL;DR: TGF‐β3 may offer a scar‐reducing therapy for acute and chronic wounds and fibrosing disorders.
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Low-Density Lipoprotein Upregulates Low-Density Lipoprotein Receptor-Related Protein Expression in Vascular Smooth Muscle Cells Possible Involvement of Sterol Regulatory Element Binding Protein-2-Dependent Mechanism
TL;DR: In situ hybridization analysis indicates that there is a higher VSMC-LRP expression in hypercholesterolemic than in normocholesterolesmic pig aortas, and indicates that L RP expression in VSMCs is upregulated by intravascular and systemic LDL.
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Mesenchymal Stem Cells in Chronic Wounds: The Spectrum from Basic to Advanced Therapy.
TL;DR: It has been demonstrated that MSCs speed up wound healing by decreasing inflammation, by promoting angiogenesis, and by decreasing scarring, but there are some potential limitations to successful MSC therapy.
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Aggregated Low-Density Lipoprotein Uptake Induces Membrane Tissue Factor Procoagulant Activity and Microparticle Release in Human Vascular Smooth Muscle Cells
TL;DR: VSMC-TF expression is upregulated by both nLDL and ag LDL, however, only agLDL engagement to LDL receptor-related protein induced cellular TF procoagulant activity and TF release by human VSMCs.