M
Martha Douglas-Escobar
Researcher at University of Florida
Publications - 21
Citations - 1248
Martha Douglas-Escobar is an academic researcher from University of Florida. The author has contributed to research in topics: Hypoxic Ischemic Encephalopathy & Neuroprotection. The author has an hindex of 11, co-authored 21 publications receiving 996 citations. Previous affiliations of Martha Douglas-Escobar include Baylor College of Medicine & University of California, San Francisco.
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Journal ArticleDOI
Hypoxic-ischemic encephalopathy: a review for the clinician.
TL;DR: The pathophysiology of HIE is now better understood, and treatment withHypothermia has become the foundation of therapy, and several neuroprotective agents offer promise when combined with hypothermia and are entering clinical trials.
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Effect of intestinal microbial ecology on the developing brain.
TL;DR: A brief review of the intestinal microbiome is provided, with a focus on new studies showing that there is an important link between the microbes that inhabit the intestinal tract and the developing brain.
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Live and heat-killed Lactobacillus rhamnosus GG: effects on proinflammatory and anti-inflammatory cytokines/chemokines in gastrostomy-fed infant rats.
TL;DR: Both live and heat-killed LGG provided by the enteral route decrease LPS-induced proinflammatory mediators and increase anti- inflammatory mediators.
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Biomarkers of hypoxic-ischemic encephalopathy in newborns
TL;DR: In this article, the most frequent source of neonatal brain injury occurs as a result of hypoxic-ischemic injury, which occurs in about 2 of 1,000 full-term infants and severe injured infants will have lifetime disabilities and neurodevelopmental delays.
Journal ArticleDOI
A Pilot Study of Novel Biomarkers in Neonates With Hypoxic-Ischemic Encephalopathy
Martha Douglas-Escobar,Cui Yang,Jeffrey Bennett,Jonathan J. Shuster,Douglas W. Theriaque,Avital Leibovici,David W. Kays,Tong Zheng,Candace Rossignol,Gerry Shaw,Michael D. Weiss +10 more
TL;DR: It is hypothesized that serum pNF-H and UCHL1 were higher in neonates with moderate-to-severe HIE than in healthy neonates and explored as diagnostic and prognostic tools in neonatal HIE.