Mary J. Pantin-Jackwood

TL;DR: Results indicate that a human influenza H1N1 virus possessing the 1918 HA and NA glycoproteins can induce severe lung inflammation consisting of AMs and neutrophils, which play a role in controlling the replication and spread of 1918 HA/NA:Tx/91 virus after intranasal infection of mice.

TL;DR: Although experimental studies using intranasal inoculation have produced infection in a variety of wild bird species, the inefficiency of contact transmission in some of them suggests they are unlikely to be a reservoir for the viruses, while others such as some wild Anseriformes, can be severely affected and could serve as a dissemination host over intermediate distances.

TL;DR: It is reinforced that enteric viruses are widespread in poultry throughout the United States, although the clinical importance of most of these viruses remains unclear.

TL;DR: Co-infection of chickens or turkeys with l NDV and LPAIV affected the replication dynamics of these viruses but did not affect clinical signs, suggesting that infection with a heterologous virus may result in temporary competition for cell receptors or competent cells for replication, most likely interferon-mediated, which decreases with time.

TL;DR: The observed differences in pathology between ducks infected at different ages is unclear and may be associated with a variety of factors including the virus strain, host immune response, host cell maturation, and capacity to support viral replication.