Matthias L. Berens

TL;DR: In this article, the authors explore the evolution of defense hormone signaling networks by combining the model plant-based knowledge about molecular components mediating phytohormone signaling and cross talk with available genome information of other plant species.

TL;DR: A genetic mechanism by which leaves of distinct ages differentially control stress-response cross-talk is identified, which balances trade-offs upon conflicting stresses at the organism level and identifies a genetic intersection among plant immunity, leaf microbiota, and abiotic stress tolerance.

TL;DR: Time-series RNA-sequencing data of Arabidopsis thaliana wild-type and combinatorial mutant plants deficient in components of the phytohormone network upon challenge with virulent or ETI-triggering avirulent strains of the foliar bacterial pathogen Pseudomonas syringae generated showed that gene coexpression network structure was highly conserved between the wild type and quadruple mutant.

TL;DR: The complexity of hormonal crosstalk in immunity and approaches currently being used to further understand this process are highlighted, as well as perspectives to engineer hormone crosStalk for enhanced pathogen resistance and overall plant fitness.

TL;DR: It is found that the phytohormones abscisic acid and jasmonate (JA), the signaling pathways of which are often exploited by pathogens, transcriptionally activate a common family of protein phosphatases that suppress immune-associated MAP kinases.