scispace - formally typeset
M

Maxwell Murphy

Researcher at University of California, Los Angeles

Publications -  6
Citations -  749

Maxwell Murphy is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Cell migration & Wound healing. The author has an hindex of 5, co-authored 6 publications receiving 427 citations.

Papers
More filters
Journal ArticleDOI

Current development of biodegradable polymeric materials for biomedical applications

TL;DR: This review outlines the current development of biodegradable natural and synthetic polymeric materials for various biomedical applications, including tissue engineering, temporary implants, wound healing, and drug delivery.
Journal ArticleDOI

Current development of silver nanoparticle preparation, investigation, and application in the field of medicine

TL;DR: The invited review covers different research areas of silver nanoparticles, including the synthesis strategies of AgNPs, antimicrobial and anti-inflammatory properties of AgnPs, osteoconductive and osteoinductive activities of AgNP-based materials, and potential toxicity ofAgNPs.
Journal ArticleDOI

Fibromodulin Enhances Angiogenesis during Cutaneous Wound Healing.

TL;DR: It is demonstrated that in addition to reducing scar formation, FMOD also promotes angiogenesis, and its potent angiogenic properties will further expand the clinical application of FMOD for cutaneous healing of poorly vascularized wounds.
Journal ArticleDOI

Fibromodulin reprogrammed cells: A novel cell source for bone regeneration.

TL;DR: In this paper, the authors further purified and significantly increased the reprogramming rate of the yield multipotent FMOD reprogrammed (FReP) cells and exposed the'molecular blueprint' of FReP cell osteogenic differentiation by gene profiling.
Journal ArticleDOI

Fibromodulin-Deficiency Alters Temporospatial Expression Patterns of Transforming Growth Factor-β Ligands and Receptors during Adult Mouse Skin Wound Healing

TL;DR: This study demonstrates that a single missing gene, FMOD, leads to conspicuous alternations in TGF-β ligand and receptor expression at all stages of wound repair in various cell types, suggesting that FMOD modulates TGF -β bioactivity in a complex way beyond simple physical binding to promote proper wound healing.