M
Md. Iqbal Alam
Researcher at Jamia Hamdard
Publications - 6
Citations - 77
Md. Iqbal Alam is an academic researcher from Jamia Hamdard. The author has contributed to research in topics: Snake venom & Venom. The author has an hindex of 3, co-authored 5 publications receiving 43 citations.
Papers
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Journal ArticleDOI
Design, synthesis and molecular docking of thiazolidinedione based benzene sulphonamide derivatives containing pyrazole core as potential anti-diabetic agents
Mohd. Javed Naim,Ozair Alam,Md. Jahangir Alam,Md. Quamrul Hassan,Nadeem Siddiqui,V.G.M. Naidu,Md. Iqbal Alam +6 more
TL;DR: 2,4-thiazolidinedione derivatives containing benzene sulphonyl group which are docked against the Peroxisome Proliferator Activated Receptor (PPARγ) target showed excellent interactions with amino acids and may be considered as promising candidates for the development of new antidiabetic agents.
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Molecular modeling and snake venom phospholipase A2 inhibition by phenolic compounds: Structure-activity relationship.
TL;DR: The anti-venom activity of the phenolic compounds which needs to be further investigated for the development of new anti-snake venom leads is confirmed.
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Naringenin mitigates behavioral alterations and provides neuroprotection against 3-nitropropinoic acid-induced Huntington's disease like symptoms in rats
TL;DR: In this paper, the neuroprotective efficacy of naringenin against 3-nitropropionic acid (3-NP)-induced neurobehavioral, biochemical and histopathological alterations in rats was investigated.
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Inhibition of snake venom induced sterile inflammation and PLA2 activity by Titanium dioxide Nanoparticles in experimental animals.
Shubhro Chakrabartty,Md. Iqbal Alam,Saumya Bhagat,Aftab Alam,Neha Dhyani,Gausal A. Khan,M. Sarwar Alam +6 more
TL;DR: The TiO2-NPs successfully neutralized the Daboia russelii venom (DRV) and Naja kaouthia venom (NKV)-induced lethal activity and the Viper venom induced haemorrhagic, coagulant and anticoagulants activities were effectively neutralized both in in-vitro and in vivo studies.
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PGE2‐induced migration of human brain endothelial cell is mediated though protein kinase A in cooperation of EP receptors
TL;DR: It is shown that PGE2 stimulated migration of HBECs in a dose‐time and matrix‐dependent manner, evaluated by the Boyden chamber assay, but other prostanoids failed to do so, and that P GE2 regulates HBEC migration via cooperation of EP2, EP3, and EP4 receptors.