M
Michael A. Muniak
Researcher at Oregon Health & Science University
Publications - 19
Citations - 784
Michael A. Muniak is an academic researcher from Oregon Health & Science University. The author has contributed to research in topics: Cochlear nucleus & Auditory system. The author has an hindex of 10, co-authored 17 publications receiving 638 citations. Previous affiliations of Michael A. Muniak include University of New South Wales & Vollum Institute.
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The Neural Coding of Stimulus Intensity: Linking the Population Response of Mechanoreceptive Afferents with Psychophysical Behavior
TL;DR: The neural code underlying the perception of stimulus intensity in the somatosensory system is investigated and it is concluded that stimulus intensity is best accounted for by the firing rate evoked in afferents located under or near the locus of stimulation, weighted by afferent type.
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The spiral ganglion: connecting the peripheral and central auditory systems
TL;DR: This report will update the progress on topics and indicate important issues still awaiting resolution about spiral ganglion neurons, including their cell biology including cytoplasmic, cytoskeletal, and membrane properties, and their capacity for plasticity and rehabilitation.
Journal Article
The neural coding of stimulus intensity: Linking the population response of mechanoreceptive afferents with psychophysical behavior (Journal of Neuroscience (October 24, 2007) (11687-11699))
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3D model of frequency representation in the cochlear nucleus of the CBA/J mouse
Michael A. Muniak,Alejandro Rivas,Karen L. Montey,Bradford J. May,Howard W. Francis,David K. Ryugo +5 more
TL;DR: This work reconstructed in 3D the trajectories of labeled auditory nerve (AN) fibers following multiunit recordings and dye injections in the anteroventral CN of the CBA/J mouse to derive a 3D quantitative map of frequency organization throughout the entire volume of the cochlear nucleus.
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Central Compensation in Auditory Brainstem after Damaging Noise Exposure.
TL;DR: The results of the current study suggest that a reorganization of excitation and inhibition in the ventral cochlear nucleus may drive hyperactivity in the central auditory system, and an increase in central gain can compensate for peripheral loss to restore some aspects of auditory function.