M
Michael Goldstein
Researcher at Beth Israel Deaconess Medical Center
Publications - 78
Citations - 4324
Michael Goldstein is an academic researcher from Beth Israel Deaconess Medical Center. The author has contributed to research in topics: Polysomnography & Cancer. The author has an hindex of 28, co-authored 67 publications receiving 3651 citations. Previous affiliations of Michael Goldstein include Hamilton College & Duke University.
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Journal ArticleDOI
Platelet Transfusion for Patients With Cancer: Clinical Practice Guidelines of the American Society of Clinical Oncology*
Charles A. Schiffer,Kenneth C. Anderson,Charles L. Bennett,Steven L. Bernstein,Linda S. Elting,Miriam Goldsmith,Michael Goldstein,Heather Hume,Jeffery J. McCullough,Rosemary E. McIntyre,Bayard L. Powell,John M. Rainey,Scott D. Rowley,Paolo Rebulla,Michael B. Troner,Alton H. Wagnon +15 more
TL;DR: The possible consequences of different approaches to the use of platelet transfusion were considered in evaluating a preference for one or another technique producing similar outcomes and cost alone was not a determining factor.
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Future Supply and Demand for Oncologists : Challenges to Assuring Access to Oncology Services
TL;DR: A multifaceted strategy will be needed to ensure that Americans have access to oncology services in 2020, as no single action will fill the likely gap between supply and demand.
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The DNA Damage Response: Implications for Tumor Responses to Radiation and Chemotherapy
TL;DR: The DDR pathways that are activated after treatment with radiation and different classes of chemotherapeutic drugs are highlighted and mechanisms determining tumor sensitivity and resistance to these agents are described.
Journal Article
The RET/PTC3 oncogene : Metastatic solid-type papillary carcinomas in murine thyroids
Daniel J. Powell,John C. Russell,Ken-ichi Nibu,Guoqi Li,Ed Rhee,Mickey Liao,Michael Goldstein,William M. Keane,Massimo Santoro,Alfredo Fusco,Jay L. Rothstein +10 more
TL;DR: A new member of this fusion oncogene family, RET/PTC3, which has been implicated in more cases of solid tumor carcinoma than PTC1 or PTC2 and predominates in radiation-induced thyroid cancer of children, was investigated in this study.
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Inactivation of the Human Papillomavirus E6 or E7 Gene in Cervical Carcinoma Cells by Using a Bacterial CRISPR/Cas RNA-Guided Endonuclease
Edward M. Kennedy,Anand V.R. Kornepati,Michael Goldstein,Hal P. Bogerd,Brigid Chiyoko Poling,Adam W. Whisnant,Michael B. Kastan,Bryan R. Cullen +7 more
TL;DR: It is demonstrated that the recently described bacterial CRISPR/Cas RNA-guided endonuclease can be reprogrammed to target and destroy the E6 or E7 gene in cervical carcinoma cells transformed by HPV, resulting in cell cycle arrest and eventual cell death.