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Michael Horowitz

Researcher at University of Adelaide

Publications -  1064
Citations -  51852

Michael Horowitz is an academic researcher from University of Adelaide. The author has contributed to research in topics: Gastric emptying & Postprandial. The author has an hindex of 112, co-authored 982 publications receiving 46952 citations. Previous affiliations of Michael Horowitz include University Hospital of Lausanne & Royal Adelaide Hospital.

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Journal ArticleDOI

Hypoglycaemia and gastric emptying.

TL;DR: The marked acceleration of gastric emptying induced by hypoglycaemia probably represents an important counter‐regulatory response to increase the rate of carbohydrate absorption.
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Effects of Sustained Treatment With Lixisenatide on Gastric Emptying and Postprandial Glucose Metabolism in Type 2 Diabetes: A Randomized Controlled Trial

TL;DR: Eight weeks’ treatment with lixisenatide is associated with sustained slowing of gastric emptying and marked reductions in postprandial glycemia and appearance of ingested glucose, and short-acting GLP-1 receptor agonists therefore potentially represent an effective long-term therapy for specifically targeting postPRandial glucose excursions.
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Radiocalcium absorption is reduced in postmenopausal women with vertebral and most types of peripheral fractures.

TL;DR: Impaired calcium absorption is particularly associated with those fractures for which osteoporosis is a significant risk factor, and age-adjusted radiocalcium absorption was significantly lower in the two groups with spinal fractures than in the controls.
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The special role of "hormonal" forms of vitamin D in the treatment of osteoporosis.

TL;DR: It is shown that the indications for treatment with vitamin D (or its 25-hydroxy metabolite) are quite distinct from the indications with what are sometimes called the “hormonal” forms of vitamin D, notably, 1-alpha and 1-25D.
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Diabetic Gastroparesis and Glycaemic Control.

TL;DR: This review summarises recent advances in the understanding of diabetic gastroparesis, with an emphasis on the current therapies that influence GE, and the bidirectional relationship between glycaemic control and GE.