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Michael L. Schilsky

Researcher at Yale University

Publications -  168
Citations -  10811

Michael L. Schilsky is an academic researcher from Yale University. The author has contributed to research in topics: Liver transplantation & Liver disease. The author has an hindex of 43, co-authored 159 publications receiving 8672 citations. Previous affiliations of Michael L. Schilsky include Cornell University & Albert Einstein College of Medicine.

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Metabolic liver disease.

TL;DR: This review of selected articles covers a wide range of subjects, from the identification of novel proteins and transport pathways to disease diagnosis and treatment of acute liver failure.
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A Multidisciplinary Approach to the Diagnosis and Management of Wilson Disease:Executive Summary of the 2022 Practice Guidance on Wilson Disease from the American Association for the Study of Liver Diseases.

TL;DR: Schilsky, Michael L *,1; Roberts, Eve A.2; Bronstein, Jeff M.3; Dahlawan, Anil4; Hamilton, James P.5; Rivard, Anne Marie6; Washington, Mary Kay7; Weiss, Karl Heinz8; Zimbrean, Paula C.9 Author Information as discussed by the authors
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A clinical assessment of Wilson disease in patients with concurrent liver disease.

TL;DR: Patients with concurrent liver diseases were diagnosed with Wilson disease at significantly older ages, presented with more liver cirrhosis, and showed greater mortality.
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COVID-19 and the Uncovering of Health Care Disparities in the United States, United Kingdom and Canada: Call to Action

TL;DR: In this article, the authors discussed the impact of the coronavirus disease 2019 pandemic on the United States, United Kingdom and Canada, highlighting the disproportionate effects on infection rates and death for certain ethnic minorities, those socioeconomically disadvantaged and living in higher density areas, and those working in health care and other essential jobs.
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HSD17B13 truncated variant is associated with a mild hepatic phenotype in Wilson's Disease

TL;DR: The results indicate that this mutation plays a role in the evolution of liver disease and likely ameliorates hepatic fibrosis and reduces the transition from copper induced hemolysis to fulminant disease in patients with WD.