T.A. Revenson, Introduction. Part 1. Overarching Frameworks and Paradigms. H. Leventhal, S. Bodnar-Deren, J.Y. Breland, J. Hash-Converse, L.A. Phillips, E.A. Leventhal, L.D. Cameron, Modeling Health and Illness Behavior: The Approach of the Common Sense Model. J.R. Jennings, V. Egizio, How Psychophysiology Contributes to Health Psychology. A. Luegey Dougall, A. Baum, Stress, Health and Illness. M.F. Scheier, C.S. Carver, G.H. Armstrong, Behavioral Self-regulation, Health and Illness. K. Glanz, M.C. Kegler, Processes of Health Behavior Change. L.S. Aiken, M.A. Gerend, K.M. Jackson, K.W. Ranby, Subjective Risk and Health Protective Behavior: Prevention and Early Detection. Part 2. Cross-cutting Issues. D. Turk, H.D. Wilson, K.S. Swanson, Psychological and Physiological Bases of Chronic Pain. J. Smyth, J.W. Pennebaker, D. Arigo, What are the Health Effects of Disclosure? T.A. Revenson, S.J. Lepore, Coping in Social Context. M.A. Hoyt, A. Stanton, Adjustment to Chronic Illness. S.I. McClelland, Measuring Sexual Quality of Life: Ten Recommendations for Health Psychologists. J Dunbar-Jacob, E. Schlenk, M. McCall, Patient Adherence to Treatment Regimens. L.M. Martire, R. Schulz, Caregiving and Care-receiving in Later Life: Health Effects and Promising Interventions. Part 3. Risk and Protective Factors. N.E. Grunberg, S. Shafer Berger, A.K. Starosciak, Tobacco Use: Psychology, Neurobiology, and Clinical Implication. R. Wing, S. Phelan, Obesity. G. Brassington, E.B. Hekler, Z. Cohen, A.C. King, Health Enhancing Physical Activity. T.W. Smith, L.C. Gallo, S. Shivpuri, A.L. Brewer, Personality and Health: Current Issues and Emerging Perspectives. C. Park, Meaning, Spirituality, and Growth: Protective and Resilience Factors in Health and Illness. C. Dunkel Schetter, M. Lobel, Pregnancy and Birth Outcomes: A Multi-level Analysis of Prenatal Maternal Stress and Birth Weight. T. Wills, M.G. Ainette, Social Networks and Social Support. M.A. Alderfer, C.M. Stanley, Health and Illness in the Context of the Family. Part 4. Macro-level and Structural Influences on Health. V.S. Helgeson, Gender and Health: A Social Psychological Perspective. J.M. Ruiz, C.C. Prather, P. Steffen, Socioeconomic Status and Health. E. Brondolo, S. Lackey, E. Love, Race and Health: Racial Disparities in Hypertension and Links Between Racism and Health. I.H. Meyer, The Health of Sexual Minorities. I. Siegler, M.F. Elias, H.B. Bosworth, Aging and Health. Part 5. Applications of Health Psychology. J.D. Betensky, R.J. Contrada, D.C. Glass, Psychosocial Factors in Cardiovascular Disease: Emotional States, Conditions, and Attributes. S.P. Newman, S.P. Hirani, J. Stygall, T. Fteropoulli, Treatment in Cardiovascular Disease. N. Schneiderman, K. Orth-Gomer, Randomized Clinical Trials: Psychosocial-behavioral Interventions for Cardiovascular Disease. L.A. Faul, P.B. Jacobsen, Psychosocial Interventions for People with Cancer. A.L. Marsland, E.A. Bachen, S. Cohen, Stress, Immunity and Susceptibility to Upper Respiratory Infectious Disease. S. Danoff-Burg, A.H. Seawell, Psychological Processes in Rheumatic Disease. M.H. Antoni, A.W. Carrico, Psychological and Bio-behavioral Processes in HIV Disease. V. Mays, R.M. Maas, J. Ricks, S.D. Cochran, HIV and African American Women in the U.S. South: A Social Determinants Approach to Population-level HIV Prevention and Intervention Efforts.

Handbook of health psychology

German Environmental Survey 1998 (GerES III): environmental pollutants in blood of the German population.

Background: Mercury (Hg) is used in gold mining to extract gold from ore by forming “amalgam”—a mixture composed of approximately equal parts mercury and gold. Approximately 15 million people, incl...

Mercury exposure and health impacts among individuals in the artisanal and small-scale gold mining community: a comprehensive review.

Mercury contamination from artisanal gold mining in Antioquia, Colombia: The world's highest per capita mercury pollution

We previously identified a functional variant of KLOTHO (termed “KL-VS”), which harbors two amino acid substitutions in complete linkage disequilibrium and is associated with reduced human longevity when in homozygosity. Klotho-deficient mice display extensive arteriosclerosis when fed a normal diet, suggesting a potent genetic predisposition. To determine whether klotho influences atherosclerotic risk in humans, we performed cross-sectional studies to assess the association between the KL-VS allele and occult coronary artery disease (CAD) in two independent samples of apparently healthy siblings of individuals with early-onset (age <60 years) CAD (SIBS-I [N=520] and SIBS-II [N=436]). Occult CAD was defined as the occurrence of a reversible perfusion defect during exercise thallium scintigraphy and/or as an abnormal result of an exercise electrocardiogram (SIBS-I, n=97; SIBS-II, n=56). In SIBS-I, the KL-VS allele conferred a relative odds of 1.90 (95% confidence interval 1.21–2.98) for occult CAD, after adjusting for familial intraclass correlations (P<.005). Logistic regression modeling, incorporating known CAD risk factors, demonstrated that the KL-VS allele is an independent risk factor (P<.019) and that the imposed risk of KL-VS allele status is influenced by modifiable risk factors. Hypertension (P<.022) and increasing high-density lipoprotein cholesterol (HDL-C) levels (P<.022) mask or reduce the risk conferred by the KL-VS allele, respectively, whereas current smoking (P<.004) increases the risk. Remarkably concordant effects of the KL-VS allele and modifying factors on the risk of occult CAD were seen in SIBS-II. These results demonstrate that the KL-VS allele is an independent risk factor for occult CAD in two independent high-risk samples. Modifiable risk factors, including hypertension, smoking status, and HDL-C level, appear to influence the risk imposed by this allele.

https://www.cell.com/ajhg/pdf/S0002-9297(07)60643-0.pdf

KLOTHO Allele Status and the Risk of Early-Onset Occult Coronary Artery Disease

Validation of self-reported smoking status by simultaneous measurement of carbon monoxide and salivary thiocyanate.

Despite a wealth of data on the neurotoxic effects of lead at the cellular and molecular levels, the reasons for its development-dependent neurotoxicity are still unclear. Here, the maturation-dependent effects of lead acetate were analyzed in immature and differentiated brain cells cultured in aggregates. Markers of general cytotoxicity as well as cell-type-specific markers of glial and neuronal cells showed that immature brain cells were more sensitive to lead than the differentiated counterparts, demonstrating that the development-dependent neurotoxicity of lead can be reproduced in aggregating brain cell cultures. After 10 days of treatment, astrocytes were found to be more affected by lead acetate than neurons in immature cultures, and microglial cells were strongly activated. Eleven days after cessation of the treatment, lead acetate caused a partial loss of astrocytes and an intense reactivity of the remaining ones. Furthermore, microglial cells expressed a macrophagic phenotype, and the loss of activity of neuron-specific enzymes was aggravated. In differentiated cultures, no reactive gliosis was found. It is hypothetized that the intense glial reactions (microgliosis and astrogliosis) observed in immature cultures contribute to the development-dependent neurotoxicity of lead.

Maturation-dependent neurotoxicity of lead acetate in vitro: implication of glial reactions.

Purpose
In Burkina Faso, gold ore is one of the main sources of income for an important part of the active population. Artisan gold miners use mercury in the extraction, a toxic metal whose human health risks are well known. The aim of the present study was to assess mercury exposure as well as to understand the exposure determinants of gold miners in Burkinabe small-scale mines.

/pdf/human-mercury-exposure-associated-with-small-scale-gold-1taf1tciu2.pdf

Human mercury exposure associated with small-scale gold mining in Burkina Faso.

The study validated the use of urinary toluene diamine (TDA) in postshift samples as an indicator of preceding 8-h exposure to toluene diisocyanate (TDI). Nine workers exposed in TDI-based polyurethane foam production were studied. Their exposure levels varied in 8-h time-averaged samples from 9.5 to 94 micrograms/m3. The urinary TDA concentrations varied from 6.5 to 31.7 micrograms/g creatinine and they were linearly related to the atmospheric TDI levels. Approximately 20% of TDI is metabolized to diamines but their specificity is remarkable to the extent that by analysis for the 2,4- and 2,6-diamino isomers an idea of the percutaneous absorption may be had.

Michèle Berode

Papers

Validation of self-reported smoking status by simultaneous measurement of carbon monoxide and salivary thiocyanate.

Maturation-dependent neurotoxicity of lead acetate in vitro: implication of glial reactions.

Human mercury exposure associated with small-scale gold mining in Burkina Faso.

Biological monitoring of occupational exposure to toluene diisocyanate

Time Trend and Determinants of Blood Lead Levels in a Swiss Population over a Transition Period (1984-1993) from Leaded to Unleaded Gasoline Use