M
Min-Zhao Huang
Researcher at University of Kansas
Publications - 4
Citations - 353
Min-Zhao Huang is an academic researcher from University of Kansas. The author has contributed to research in topics: Osteocalcin & GPRC6A. The author has an hindex of 4, co-authored 4 publications receiving 326 citations. Previous affiliations of Min-Zhao Huang include University of Tennessee Health Science Center.
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Journal ArticleDOI
GPRC6A Null Mice Exhibit Osteopenia, Feminization and Metabolic Syndrome
Min Pi,Ling Chen,Min-Zhao Huang,Wenyu Zhu,Brian Ringhofer,Junming Luo,Lane K. Christenson,Benyi Li,Jianghong Zhang,P. David Jackson,Pieter W. Faber,Kurt R. Brunden,John J. Harrington,L. Darryl Quarles +13 more
TL;DR: The overall function of GPRC6A may be to coordinate the anabolic responses of multiple tissues through the sensing of extracellular amino acids, osteocalcin and divalent cations.
Journal ArticleDOI
Impaired Osteoblast Function in GPRC6A Null Mice
Min Pi,Lishu Zhang,Lishu Zhang,Shu-Feng Lei,Min-Zhao Huang,Wenyu Zhu,Jianghong Zhang,Hui Shen,Hong-Wen Deng,Hong-Wen Deng,L. Darryl Quarles +10 more
TL;DR: Data indicate that GRPC6A directly participates in the regulation of osteoblast‐mediated bone mineralization and may mediate the anabolic effects of extracellular amino acids, osteocalcin, and divalent cations in bone.
Journal ArticleDOI
Embelin suppresses growth of human pancreatic cancer xenografts, and pancreatic cancer cells isolated from KrasG12D mice by inhibiting Akt and Sonic hedgehog pathways.
Min-Zhao Huang,Su-Ni Tang,Ghanshyam Upadhyay,Justin L. Marsh,Christopher P. Jackman,Sharmila Shankar,Rakesh K. Srivastava +6 more
TL;DR: The data suggest that embelin can inhibit pancreatic cancer growth, angiogenesis and metastasis by suppressing Akt and Shh pathways, and can be developed for the treatment and/or prevention of Pancreatic cancer.
Journal ArticleDOI
Parathyroid-specific interaction of the calcium-sensing receptor and Gαq
TL;DR: The feasibility of selectively targeting the parathyroid gland to investigate signaling mechanisms downstream of the calcium receptor is demonstrated.