M
Mingzhou Liu
Researcher at Zhengzhou University
Publications - 4
Citations - 73
Mingzhou Liu is an academic researcher from Zhengzhou University. The author has contributed to research in topics: Medicine & Neurite. The author has an hindex of 1, co-authored 2 publications receiving 37 citations.
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Journal ArticleDOI
Long Non-coding RNA MALAT1 Inhibits Neuron Apoptosis and Neuroinflammation While Stimulates Neurite Outgrowth and Its Correlation With MiR-125b Mediates PTGS2, CDK5 and FOXQ1 in Alzheimer's Disease.
TL;DR: Lnc-MALAT1 might be interacted with miR-125b to inhibit neuron apoptosis and inflammation while promote neurite outgrowth in AD.
Journal ArticleDOI
Detection of sivelestat and its metabolite in small volumes of plasma from Chinese ALI/ARDS patients with SIRS via high-throughput UPLC-MS/MS: A pharmacokinetic study.
Mingzhou Liu,Jing Zhang,Lingfang Dong,Wenhua Xue,Qilin He,Wenzhong Liang,Xing Liu,Jingying Zhang,Li Gu,Yinghua Feng,Jie Yang,Haibo Wang,Yaqin Wang,Kun Li,Yuanlong Li,Weiqin Kong,Xiaojian Zhang,Mengying Yao,Kai Wang,Peizhi Ma,Wei Zhang +20 more
TL;DR: In this paper, a 96-well plate-based protein precipitation strategy was combined with ultra-performance liquid chromatography electrospray ionization tandem mass spectrometry (UPLC-MS/MS) to assess the pharmacokinetic (PK) properties of sivelestat and its metabolite XW-IMP-A in samples of plasma from ALI/ARDS patients with SIRS.
Journal Article
A novel cuproptosis-related signature predicts prognosis and immunotherapy efficacy in lung adenocarcinoma.
TL;DR: In this article , a ten-gene signature associated with apoptosis in cuproptosis was built and demonstrated the ability to predict response to LUAD immunotherapy by applying TMB, immune relevant signatures, and TIDE.
Journal ArticleDOI
GPD1 inhibits the carcinogenesis of breast cancer through increasing PI3K/AKT-mediated lipid metabolism signaling pathway
TL;DR: Wang et al. as mentioned in this paper found that GPD1 overexpression activated the lipid synthesis pathway and PI3K/AKT signaling pathway and inhibited the ability of proliferation, migration, and invasion in GPD 1 overexpressive breast cancer cells by CCK-8, wound healing, and Transwell assays.