Miren Iturriza Gómara

TL;DR: Molecular characterization of the SG-defining region of VP6 provided evidence for independent segregation of the rotavirus genes encoding VP4, VP6, and VP7, and revealed two clusters, or genogroups.

TL;DR: Early infection and frequent reinfection in a locale with high viral diversity resulted in lower protection than has been reported elsewhere, providing a possible explanation why rotavirus vaccines have had lower-than-expected efficacy in Asia and Africa.

TL;DR: The low incidence of uncommon strains would suggest that transmission, or at least the establishment of an animal rotavirus or a human/animal reassortant virus in the human population does not happen with any great frequency.

TL;DR: Characterization of the genes encoding VP7, VP4, VP6, and NSP4 of these strains revealed high sequence homology with the corresponding genes of the asymptomatic neonatal strain I321, which in turn is very closely related to bovine G10P[11] strains circulating in India.

TL;DR: The deduced amino acid sequences of the antigenic regions A, B, and C of VP7 revealed that a substitution at position 96 (Asp→Asn) correlated with the change in ability to serotype these G2 strains.