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Mohammed Amrani

Researcher at Harefield Hospital

Publications -  34
Citations -  1242

Mohammed Amrani is an academic researcher from Harefield Hospital. The author has contributed to research in topics: Transplantation & Ventricular assist device. The author has an hindex of 14, co-authored 34 publications receiving 1182 citations. Previous affiliations of Mohammed Amrani include University of Louisville & National Institutes of Health.

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Reversal of Severe Heart Failure With a Continuous-Flow Left Ventricular Assist Device and Pharmacological Therapy A Prospective Study

TL;DR: In this paper, the authors reported the results of a prospective study of 20 patients who received a combination of angiotensin-converting enzymes, β-blockers, angiotenin II inhibitors, and aldosterone antagonists followed by the β2-agonist clenbuterol and were regularly tested with the pump at low speed.
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Searching for the second best graft for coronary artery bypass surgery: a network meta-analysis of randomized controlled trials

TL;DR: The present network meta-analysis consistently demonstrated an angiographic superiority of RIMA and RA over RA and right internal mammary artery over saphenous vein grafting.
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Randomized Controlled Study Investigating the Effect of Biatrial Pacing in Prevention of Atrial Fibrillation After Coronary Artery Bypass Grafting

TL;DR: Biatrial pacing after CABG significantly decreases the incidence of AF and is associated with reduced postoperative complications and a trend toward reduced ICU and hospital stay.
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Bridge to decision using the Levitronix CentriMag short-term ventricular assist device.

TL;DR: The Levitronix device is effective in rescuing critically ill "moribund" patients and can provide an opportunity for low-cost support and optimization of their condition prior to deciding whether a more expensive device should be placed or if transplantation should be undertaken.
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Role of basal release of nitric oxide on coronary flow and mechanical performance of the isolated rat heart.

TL;DR: Evidence is provided that NO plays a crucial role in the maintenance of basal coronary flow and appears to be essential for sustaining mechanical activity.