M
Mohd. Salman
Researcher at Hamdard University
Publications - 6
Citations - 150
Mohd. Salman is an academic researcher from Hamdard University. The author has contributed to research in topics: Oxidative stress & Neurotoxicity. The author has an hindex of 4, co-authored 5 publications receiving 108 citations.
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Journal ArticleDOI
Neuromodulatory effects of hesperidin in mitigating oxidative stress in streptozotocin induced diabetes.
Mohammad Ashafaq,Laxmi Varshney,Mohammad Haaris Ajmal Khan,Mohd. Salman,Mehar Naseem,Saima Wajid,Suhel Parvez +6 more
TL;DR: It is demonstrated that hesperidin exhibits potent antioxidant and neuroprotective effects on the brain tissue against the diabetic oxidative damage in STZ-induced rodent model and significantly attenuated the altered levels of oxidative stress and neurotoxicity biomarkers.
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Propiconazole induced toxicological alterations in brain of freshwater fish Channa punctata Bloch
TL;DR: The results point towards the potential neurotoxicity in the fish caused by PCZ exposure but the application of these findings will need more detailed study before they can be established as special biomarkers for toxicity monitoring the aquatic environment.
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Melatonin pre-treatment mitigates SHSY-5Y cells against oxaliplatin induced mitochondrial stress and apoptotic cell death
Mohammad Waseem,Upasana Sahu,Mohd. Salman,Arnab Choudhury,Sudeshna Kar,Heena Tabassum,Suhel Parvez +6 more
TL;DR: A role for melatonin is suggested in reducing Oxa induced neurotoxicity and further studies exploring melatonin’s protective effects may prove successful in eliciting pathways to further alter the neurotoxic pathways of platinum compounds in cancer treatment.
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Tannic acid alleviates lead acetate-induced neurochemical perturbations in rat brain
TL;DR: The data suggest that LA induces oxidative stress and supplementation of TA has a powerful antioxidant effect, and it protected rat brain from poisonous effect of LA exposure in experimental rat.
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Tempol (4-hydroxy tempo) protects mice from cisplatin-induced acute kidney injury via modulation of expression of aquaporins and kidney injury molecule-1.
Mohd Amir Afjal,Poonam Goswami,Shahzad Ahmad,Sadaf Dabeer,Juheb Akhter,Mohd. Salman,Anuradha Mangla,Sheikh Raisuddin +7 more
TL;DR: Experimental evidence is provided that tempol resolved urinary concentration defect by the restoration of AQP, AVP and KIM-1 levels indicating a potential use of tempol in ameliorating the AKI in cancer patients under the treatment with CP.