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Monika Maurhofer

Researcher at ETH Zurich

Publications -  82
Citations -  5994

Monika Maurhofer is an academic researcher from ETH Zurich. The author has contributed to research in topics: Pseudomonas fluorescens & Pseudomonas protegens. The author has an hindex of 35, co-authored 77 publications receiving 5413 citations. Previous affiliations of Monika Maurhofer include École Polytechnique Fédérale de Lausanne.

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Suppression of Root Diseases by Pseudomonas fluorescens CHA0: Importance of the Bacterial Secondary Metabolite 2,4-Diacetylphloroglucinol

TL;DR: Pseudomonas fluorescens strain CHAO suppresses Thielaviopsis basicola-induced black root rot of tobacco and Gaeumannomyces graminis var. tritici-induced take-all of wheat.
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Induction of systemic resistance of tobacco to tobacco necrosis virus by the root-colonizing Pseudomonas fluorescens strain CHA0: influence of the gacA gene and of pyoverdine production

TL;DR: Plants tested showed resistance in leaves to infection with tobacco necrosis virus to the same extent as plants previously immunized with TNY (induced resistance control) and Pseudomonas fluorescens strain CHA0, which suppresses various plant diseases caused by soilborne pathogens also can restrict leaf disease.
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Global control in Pseudomonas fluorescens mediating antibiotic synthesis and suppression of black root rot of tobacco.

TL;DR: The gacA mutants of strain CHA0 have a drastically reduced ability to suppress black root rot under gnotobiotic conditions, supporting the previous observations that the antibiotic Phl and HCN individually contribute to the suppression of blackroot rot.
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Salicylic Acid Biosynthetic Genes Expressed in Pseudomonas fluorescens Strain P3 Improve the Induction of Systemic Resistance in Tobacco Against Tobacco Necrosis Virus.

TL;DR: Introduction of pchBA into CHA0 increased the production of salicylic acid in vitro and in the rhizosphere of tobacco, but did not improve the ability ofCHA0 to induce systemic resistance in tobacco.
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Autoinduction of 2,4-diacetylphloroglucinol biosynthesis in the biocontrol agent Pseudomonas fluorescens CHA0 and repression by the bacterial metabolites salicylate and pyoluteorin.

TL;DR: Results show that autoinduction of 2,4-DAPG biosynthesis can be countered by certain bacterial (and fungal) metabolites, which depends on phlF function, and may help P. fluorescens to produce homeostatically balanced amounts of extracellular metabolites.