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Monika Sundl

Researcher at Medical University of Graz

Publications -  11
Citations -  392

Monika Sundl is an academic researcher from Medical University of Graz. The author has contributed to research in topics: Trophoblast & Intervillous space. The author has an hindex of 7, co-authored 11 publications receiving 290 citations. Previous affiliations of Monika Sundl include University of Graz.

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Extravillous trophoblasts invade more than uterine arteries: evidence for the invasion of uterine veins

TL;DR: It is suggested that invasion of EVTs into uterine veins is responsible the draining of waste and blood plasma from the intervillous space during the first trimester of pregnancy.
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The trophoblast plug during early pregnancy: a deeper insight

TL;DR: This study demonstrates for the first time serial cross sections through a trophoblast plug in a first trimester placental bed specimen and gives evidence of the presence of MMP-1 in plugs of invaded spiral arteries.
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Evidence from the very beginning: endoglandular trophoblasts penetrate and replace uterine glands in situ and in vitro

TL;DR: This study further strengthens the hypothesis of histiotrophic nutrition of the embryo prior to the establishment of the maternal blood flow toward the placenta by using a previously established three-dimensional co-culture in vitro model system of first trimester villous explants with non-invaded decidua parietalis.
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Expression of serum amyloid A transcripts in human bone tissues, differentiated osteoblast-like stem cells and human osteosarcoma cell lines.

TL;DR: It is shown that SAA1 (coding for the major SAA isoform) but not SAA2 transcripts are expressed in human trabecular and cortical bone fractions and bone marrow and evidence that human osteoblast‐like cells of tumor origin express SAF‐1 under basal conditions.
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Expression of serum amyloid A transcripts in human trophoblast and fetal-derived trophoblast-like choriocarcinoma cells.

TL;DR: First trimester trophoblast and JAR/Jeg‐3 cells further express the human homolog of SAA‐activating factor‐1, a transcription factor involved in cytokine‐mediated induction of A‐SAA genes, suggesting that A-SAA plays a role during early fetal development.