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Morteza Masoumi

Researcher at Pasteur Institute of Iran

Publications -  30
Citations -  296

Morteza Masoumi is an academic researcher from Pasteur Institute of Iran. The author has contributed to research in topics: Mycobacterium tuberculosis & Tuberculosis. The author has an hindex of 8, co-authored 24 publications receiving 178 citations. Previous affiliations of Morteza Masoumi include Islamic Azad University.

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High Prevalence of Bedaquiline Resistance in Treatment-Naive Tuberculosis Patients and Verapamil Effectiveness.

TL;DR: It was concluded that efflux pumps play a pivotal role in intrinsicBDQ resistance; therefore, the potential of verapamil as an adjunctive therapy to combat BDQ resistance should be investigated.
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Scrutinizing the drug resistance mechanism of multi- and extensively-drug resistant Mycobacterium tuberculosis: mutations versus efflux pumps.

TL;DR: Results clearly showed that efflux systems, besides spontaneous mutations, play a role in the development of INH/RIF resistance and although VP was effective in reducing the expression of some efflux pumps, it was not very successful at the phenotypic level.
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Evaluation of the impact of polyclonal infection and heteroresistance on treatment of tuberculosis patients.

TL;DR: It is concluded that mixed infections, have a negative impact on treatment of TB patients especially when co-infecting M. tuberculosis strains display heteroresistance.
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HSP65-PRA identification of non-tuberculosis mycobacteria from 4892 samples suspicious for mycobacterial infections.

TL;DR: Evaluation of antibiotic resistance and PCR-restriction fragment length polymorphism analysis (PRA) of the hsp65 gene showed that PRA, in comparison with classical methods, is rapid and accurate enough for the identification of mycobacterial species from LJ medium, and it was found that in Iran the authors have a highly diverse population of NTM isolates among patients suspected of having TB.
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The anti-inflammatory effects of Akkermansia muciniphila and its derivates in HFD/CCL4-induced murine model of liver injury

TL;DR: In this article , the effects of live and pasteurized A. muciniphila and its extra cellular vesicles (EVs) on inflammatory markers involved in liver fibrosis in a mouse model of a high-fat diet (HFD)/carbon tetrachloride (CCl4)-induced liver injury was evaluated.