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Muslum Gok

Researcher at Hacettepe University

Publications -  12
Citations -  121

Muslum Gok is an academic researcher from Hacettepe University. The author has contributed to research in topics: Butyrylcholinesterase & Diabetes mellitus. The author has an hindex of 5, co-authored 9 publications receiving 64 citations. Previous affiliations of Muslum Gok include Muğla University.

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Impact of the Di(2-Ethylhexyl) Phthalate Administration on Trace Element and Mineral Levels in Relation of Kidney and Liver Damage in Rats

TL;DR: Investigation of trace element and mineral levels in relation of kidney and liver damage in DEHP-administered rats indicated that sodium (Na), calcium (Ca), potassium (K), lithium (Li), rubidium (Rb) and cesium (Cs) levels significantly decreased, however iron (Fe) and selenium (Se) concentrations significantly increased inDEHP- Administered groups compared to the control in the serum samples.
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Data the DEHP induced changes on the trace element and mineral levels in the brain and testis tissues of rats

TL;DR: The data revealed that, antioxidant enzyme activities in the brain and testis samples were statistically insignificant in the DEHP administered groups compared to the control group except 400 mg/kg/day DEHP dose group in thetestis samples.
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Composite nanofibers incorporating alpha lipoic acid and atorvastatin provide neuroprotection after peripheral nerve injury in rats.

TL;DR: The neuroprotection potential of these novel nanocomposite fiber formulations has been demonstrated after local implantation of composite nanofiber sheets incorporating ALA and ATR, which contributed to the recovery of the motor and sensory function and nerve regeneration in a rat sciatic nerve crush injury model.
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Long-term treatment with a beta-blocker timolol attenuates renal-damage in diabetic rats via enhancing kidney antioxidant-defense system

TL;DR: Timolol-treatment of streptozotocin-induced diabetic rats has advantage to prevent hyperglycemia-induced renal-damage via enhancing the depressed antioxidant defense in the kidney, and β-blocker therapy of diabetics seems to be a new therapeutic approach against hyperglyCEmia- induced kidney damage in diabetic patients.