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Mustafa Cekmen

Researcher at Kocaeli University

Publications -  107
Citations -  2772

Mustafa Cekmen is an academic researcher from Kocaeli University. The author has contributed to research in topics: Nitric oxide & Malondialdehyde. The author has an hindex of 28, co-authored 102 publications receiving 2542 citations. Previous affiliations of Mustafa Cekmen include İnönü University & University of Gaziantep.

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Serum levels of TNF-alpha, sIL-2R, IL-6, and IL-8 are increased and associated with elevated lipid peroxidation in patients with Behçet's disease.

TL;DR: High levels of cytokines, chemokines and lipid peroxidation in the active period of Behçet's disease indicate the activation of immune system in BD and therefore tissue damage in such patients.
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Protective effect of curcumin in cisplatin-induced oxidative injury in rat testis: mitogen-activated protein kinase and nuclear factor-kappa B signaling pathways

TL;DR: MAPK and NF-kB activation have a significant role in CIS-induced testicular toxicity and CMN has a strong potential for use as a therapeutic adjuvant in CIS gonadotoxicity.
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Potential chemoprotective effect of melatonin in cyclophosphamide- and cisplatin-induced testicular damage in rats.

TL;DR: In this paper, the effect of melatonin on cyclophosphamide and cisplatin-induced testicular toxicity with use of sperm parameters and biochemical and histopathologic approaches was evaluated.
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Atorvastatin Prevents Gentamicin-Induced Renal Damage in Rats through the Inhibition of p38-MAPK and NF-kB Pathways

TL;DR: In this article, Gentamicin (GM) is still considered an important antibiotic against life-threatening, gram-negative bacterial infections despite its known nephrotoxic effects, and it is considered to be an effective antibiotic against various types of infections.
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Nitric oxide and lipid peroxidation are increased and associated with decreased antioxidant enzyme activities in patients with age-related macular degeneration.

TL;DR: It is demonstrated for the first time that NO, the most abundant free-radical in the body, might be implicated in the pathophysiology of ARMD in association with decreased antioxidant enzymes and increased lipid peroxidation status.