N
Nephi Stella
Researcher at University of Lausanne
Publications - 4
Citations - 819
Nephi Stella is an academic researcher from University of Lausanne. The author has contributed to research in topics: Neurotransmission & Glutamatergic. The author has an hindex of 4, co-authored 4 publications receiving 768 citations.
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Journal ArticleDOI
Evidence Supporting the Existence of an Activity-Dependent Astrocyte-Neuron Lactate Shuttle
Luc Pellerin,Giovanni Pellegri,Philippe G. Bittar,Yves Charnay,Constantin Bouras,Jean-Luc Martin,Nephi Stella,Pierre J. Magistretti +7 more
TL;DR: In this paper, the presence of mRNA encoding for two monocarboxylate transporters, MCT1 and MCT2, was found to be consistent with the existence of an activitydependent astrocyte-neuron lactate shuttle for the supply of energy substrates to neurons.
Journal ArticleDOI
Arachidonic acid stimulates glucose uptake in cerebral cortical astrocytes.
TL;DR: The effects of NE reported here further stress the notion that certain neurotransmitters may play a role in the regulation of energy metabolism in the cerebral cortex and point at astrocytes as the likely targets of such metabolic effects.
Journal ArticleDOI
Modulation of the glutamate-evoked release of arachidonic acid from mouse cortical neurons: involvement of a pH-sensitive membrane phospholipase A2
TL;DR: Results indicate that the glutamate-evoked release of 3H-AA may be mediated by the pH- sensitive mPLA2, and reveals a possible molecular mechanism whereby glutamate can modulate its own signalling efficacy in a pH-dependent manner by regulating the release of AA.
Journal ArticleDOI
Vasoactive Intestinal Peptide (VIP) and Pituitary Adenylate Cyclase-activating Polypeptide (PACAP) Potentiate the Glutamate-evoked Release of Arachidonic Acid from Mouse Cortical Neurons EVIDENCE FOR A cAMP-INDEPENDENT MECHANISM
TL;DR: Results indicate that the release of AA evoked by glutamate is potentiated by vasoactive intestinal peptide and pituitary adenylate cyclase-activating polypeptide (PACAP), and results indicate that this potentiating mechanism mediated by PACAP I receptor acts at a level downstream of the glutamate receptor-mediated calcium influx.