N
Nicholas Pocock
Researcher at St. Vincent's Health System
Publications - 105
Citations - 6962
Nicholas Pocock is an academic researcher from St. Vincent's Health System. The author has contributed to research in topics: Bone mineral & Osteoporosis. The author has an hindex of 37, co-authored 104 publications receiving 6765 citations. Previous affiliations of Nicholas Pocock include University of Melbourne & Garvan Institute of Medical Research.
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Journal ArticleDOI
Genetic determinants of bone mass in adults. A twin study.
Nicholas Pocock,John A. Eisman,John L. Hopper,Michael G. Yeates,Philip N. Sambrook,Stefan Eberl +5 more
TL;DR: The lesser genetic contribution to proximal femur and distal forearm bone mass compared with the spine suggests that environmental factors are of greater importance in the aetiology of osteopenia of the hip and wrist.
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Prevention of corticosteroid osteoporosis. A comparison of calcium, calcitriol, and calcitonin.
Philip N. Sambrook,J Birmingham,Paul Kelly,Susan Kempler,Tuan V. Nguyen,Nicholas Pocock,John A. Eisman +6 more
TL;DR: Calcitriol and calcium, used prophylactically with or without calcitonin, prevent corticosteroid-induced bone loss in the lumbar spine.
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Muscle strength, physical fitness, and weight but not age predict femoral neck bone mass.
Nicholas Pocock,Nicholas Pocock,John A. Eisman,Tom H. Gwinn,Philip N. Sambrook,Paul J. Kelly,Judith Freund,Michael G. Yeates +7 more
TL;DR: The relative importance of muscle strength, physical fitness, and body mass index (BMI) in addition to age in the determination of proximal femoral BMD in 73 healthy female volunteers age 20–75 years is examined.
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Physical fitness is a major determinant of femoral neck and lumbar spine bone mineral density.
TL;DR: The data suggest that increased physical fitness may increase bone mass at the sites of clinically important fractures in osteoporosis.
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Genetic factors in bone turnover.
Paul J. Kelly,John L. Hopper,Gregory T. Macaskill,Nicholas Pocock,Philip N. Sambrook,John A. Eisman +5 more
TL;DR: The data indicate that circulating osteocalcin, and therefore bone formation, is strongly genetically determined and suggest at least one of the mechanisms of the genetic effect on bone mass relates to the regulation of bone turnover.