O
Omid Gholami
Researcher at Shahid Beheshti University of Medical Sciences and Health Services
Publications - 23
Citations - 204
Omid Gholami is an academic researcher from Shahid Beheshti University of Medical Sciences and Health Services. The author has contributed to research in topics: Medicine & Jurkat cells. The author has an hindex of 7, co-authored 17 publications receiving 140 citations. Previous affiliations of Omid Gholami include Mashhad University of Medical Sciences.
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Journal ArticleDOI
Umbelliprenin Induces Apoptosis in CLL Cell Lines
TL;DR: The hypothesis that oral administration of umbelliprenin in the form of foods or folk medicines containing this coumarin, might enhance protection against the development of CLL in man with little side effects is supported.
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Umbelliprenin from Ferula szowitsiana Activates both Intrinsic and Extrinsic Pathways of Apoptosis in Jurkat T-CLL cell line.
TL;DR: It is shown that umbelliprenin activates intrinsic and extrinsic pathways of apoptosis by the activation of caspase-8 and -9 respectively and inhibition of Bcl-2 was also shown.
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The Cytotoxic Effects of Thymol as the Major Component of Trachyspermum ammi on Breast Cancer (MCF-7) Cells
Hasan Rezaei Seresht,Bushra Jabbar Al-Badry,Aseel Kamil Mohammed Al-mosawi,Omid Gholami,Hamid Cheshomi +4 more
TL;DR: Thymol can induce the apoptosis process in MCF-7, and hence it can be considered an anticancer agent in the future.
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Opium Addiction Increases Interleukin 1 Receptor Antagonist (IL-1Ra) in the Coronary Artery Disease Patients
Habibollah Saadat,Seyed Ali Ziai,Maryam Ghanemnia,Mohammad Hasan Namazi,Morteza Safi,Hosein Vakili,Ali Dabbagh,Omid Gholami +7 more
TL;DR: In this article, the authors evaluated the IL-1Ra levels of 30 patients with three-vessel coronary artery disease, ejection fraction of more than 35% and evaluated their changes after prognostic treadmill test in 15 opium addicted and 15 non-addicted patients.
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Mcl-1 is up regulated by prenylated coumarin, umbelliprenin in jurkat cells
TL;DR: Umbelliprenin treatment modulates Mcl-1 expression at both the transcriptional and posttranslational levels, where its expression is significantly associated with a failure to achieve complete remission following cytotoxic therapy.