O
Omran Karmach
Researcher at University of California, Riverside
Publications - 5
Citations - 139
Omran Karmach is an academic researcher from University of California, Riverside. The author has contributed to research in topics: Gene & Downregulation and upregulation. The author has an hindex of 2, co-authored 3 publications receiving 98 citations.
Papers
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Journal ArticleDOI
Red-shifted luciferase–luciferin pairs for enhanced bioluminescence imaging
Hsien-Wei Yeh,Omran Karmach,Ao Ji,David Carter,Manuela Martins-Green,Hui-wang Ai,Hui-wang Ai +6 more
TL;DR: The development of red-shifted luciferins based on synthetic coelenterazine analogs and corresponding mutants of NanoLuc that enable bright bioluminescence are described and one pair showed superior in vitro and in vivo sensitivity over commonly used biolumeinescence reporters is adapted.
Journal ArticleDOI
A membrane-activatable near-infrared fluorescent probe with ultra-photostability for mitochondrial membrane potentials
TL;DR: A photostable near-infrared (NIR) fluorescent dye for monitoring MMP, named NIMAP, is non-fluorescent in aqueous solution and can be activated by cell membranes, providing high fluorescence contrast and low background fluorescence.
Journal ArticleDOI
Embryonic Exposure to Cigarette Smoke Extract Impedes Skeletal Development and Evokes Craniofacial Defects in Zebrafish
TL;DR: Results in a model system often used to detect embryonic malformations imply that exposure of a woman to secondhand smoke while pregnant may lead to mineralization issues in the skeleton of her newborn, ultimately adding a direct in utero association to the increased fracture risk observed in children of mothers exposed to cigarette smoke.
Journal ArticleDOI
Hyperglycemia Induced Oxidative Stress Alters the FOXO/AMPK Pathway Which Leads to Loss of Pluripotency
TL;DR: The role of F-box protein 11 (FBXO11)-mediated ubiquitination and degradation in the degradation of BCL6 was investigated in this paper , where GC-specific FBXO 11 inactivation was associated with the development of lymphoproliferative disorders in mice.