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Pamir Atagündüz

Researcher at Marmara University

Publications -  76
Citations -  1825

Pamir Atagündüz is an academic researcher from Marmara University. The author has contributed to research in topics: Medicine & Ankylosing spondylitis. The author has an hindex of 20, co-authored 66 publications receiving 1375 citations. Previous affiliations of Pamir Atagündüz include Marche Polytechnic University & Charité.

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Cytokine storm in COVID-19: pathogenesis and overview of anti-inflammatory agents used in treatment.

TL;DR: Early recognition and appropriate treatment of immunologic complications will decrease the morbidity and mortality in COVID-19 infection, which requires the collaboration of infectious disease, lung, and intensive care unit specialists with other experts such as immunologists, rheumatologists, and hematologists.
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MEFV mutations are increased in Behçet's disease (BD) and are associated with vascular involvement

TL;DR: MEFV mutations, originally linked to FMF, may act as a genetic susceptibility factor for other inflammatory disorders such as vascular BD, and is investigated in patients from Turkey.
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HLA-B27-restricted CD8+ T cell response to cartilage-derived self peptides in ankylosing spondylitis.

TL;DR: The findings suggest that cartilage-directed cellular autoimmunity might play an important role in joint-specific tissue damage in patients with AS and suggest an HLA-B27-restricted immune response.
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Monitoring Achilles enthesitis in ankylosing spondylitis during TNF-α antagonist therapy: an ultrasound study

TL;DR: Subclinical Achilles enthesitis, detected only with GS US, is present in a subset of AS patients and a significant improvement can be demonstrated after 2 months of TNF-alpha antagonist therapy.
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Hemophagocytic lymphohistiocytosis: a review inspired by the COVID-19 pandemic.

TL;DR: The pathogenesis and the clinical picture of HLH, and its severe complication, the cytokine storm, is reviewed with a special emphasis on the developed classification criteria sets for rheumatologists, since COVID-19 infection has clinical symptoms resembling those of the common r heumatologic conditions and possibly triggers HLH.