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Patricia P. Long

Researcher at Johns Hopkins University

Publications -  21
Citations -  1066

Patricia P. Long is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Karyotype & Cytogenetics. The author has an hindex of 17, co-authored 21 publications receiving 1047 citations. Previous affiliations of Patricia P. Long include Johns Hopkins University School of Medicine.

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Journal Article

Fluorescence in situ hybridization for the Y-chromosome can be used to detect cells of recipient origin in allografted hearts following cardiac transplantation.

TL;DR: It is demonstrated that, when the recipient and donor are of the opposite sex, fluorescence in situ hybridization for the Y-chromosome can be used to detect graft chimerism in transplanted hearts.
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Consistent chromosome abnormalities in adenocarcinoma of the pancreas

TL;DR: To the knowledge, these represent the first primary specimens of pancreatic adenocarcinoma with cytogenetic evidence of gene amplification and chromosomal arms most frequently involved were 1p; 6q (11); 7q and 17p (9 each); and 1q, 3p, 11p, and 19q (8 each).
Journal Article

Consistent Chromosome Abnormalities in Adenocarcinoma of the Pancreas

TL;DR: In this paper, the authors studied 62 primary pancreatic adenocarcinomas obtained from surgical resections using classical cytogenetics and fluorescent in situ hybridization methods and found that the most frequent whole chromosomal gains were chromosomes 20 (eight tumors) and 7 (seven tumors).
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Chromosome abnormalities in low-grade central nervous system tumors.

TL;DR: The cytogenetics of low-grade CNS tumors differ from higher grade gliomas in that most low- grade tumors show little deviation from the normal karyotype.
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RANBP2 and CLTC are involved in ALK rearrangements in inflammatory myofibroblastic tumors

TL;DR: FISH evaluation of nine other IMTs identified CLTC as the fusion partner in one additional case, but RANBP2 was not involved in the remaining eight IMTs, suggesting that the variant partners involved in ALK rearrangements in IMTs are diverse.