P
Patrick Rebernik
Researcher at Medical University of Vienna
Publications - 13
Citations - 269
Patrick Rebernik is an academic researcher from Medical University of Vienna. The author has contributed to research in topics: Spermine & Polyamine. The author has an hindex of 8, co-authored 13 publications receiving 196 citations. Previous affiliations of Patrick Rebernik include University of Vienna.
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Journal ArticleDOI
Molecular design of hypothalamus development
Roman A. Romanov,Roman A. Romanov,Evgenii O. Tretiakov,Maria Eleni Kastriti,Maria Eleni Kastriti,Maja Zupancic,Martin Häring,Solomiia Korchynska,Konstantin Popadin,Konstantin Popadin,Marco Benevento,Patrick Rebernik,Francois Lallemend,Katsuhiko Nishimori,Frédéric Clotman,William Andrews,John G. Parnavelas,Matthias Farlik,Matthias Farlik,Christoph Bock,Christoph Bock,Igor Adameyko,Igor Adameyko,Tomas Hökfelt,Erik Keimpema,Tibor Harkany,Tibor Harkany +26 more
TL;DR: Overall, molecular principles that shape the developmental architecture of the hypothalamus are identified and it is shown how neuronal heterogeneity is transformed into a multimodal neural unit to provide virtually infinite adaptive potential throughout life.
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NMDA receptor affinities of 1,2-diphenylethylamine and 1-(1,2-diphenylethyl)piperidine enantiomers and of related compounds.
TL;DR: The synthesis of the dicyclohexyl analogues of DPEA and related compounds were tested as inhibitors of [(3)H]MK-801 binding to rat brain membranes and exhibited stereospecific sensitivity to the modulator spermine.
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Zinc regulates the dopamine transporter in a membrane potential and chloride dependent manner.
TL;DR: This study represents the first evidence that DAT regulation by Zn(2+) is profoundly modulated by the membrane potential and chloride.
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Zn2+ modulates currents generated by the dopamine transporter: parallel effects on amphetamine-induced charge transfer and release.
TL;DR: The stimulatory action of Zn(2+) on release and inward current was in contrast to its inhibitory action on dopamine uptake, which supports a release mechanism of amphetamine different from facilitated exchange diffusion but involving ion fluxes through the dopamine transporter.
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The noradrenaline transporter as site of action for the anti-Parkinson drug amantadine
TL;DR: Findings and differences with memantine, which had been reported as less efficient antiparkinsonian drug than amantadine but in the authors' hands was significantly more potent at the NMDA-receptor, suggest contributions from a noradrenergic mechanism in the antiparksinsonian action of amantADine.