P
Pawel Zmora
Researcher at Max Planck Society
Publications - 26
Citations - 943
Pawel Zmora is an academic researcher from Max Planck Society. The author has contributed to research in topics: Rumen & Proteases. The author has an hindex of 15, co-authored 20 publications receiving 774 citations. Previous affiliations of Pawel Zmora include German Primate Center.
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Journal ArticleDOI
Proteolytic activation of the SARS-coronavirus spike protein: cutting enzymes at the cutting edge of antiviral research.
TL;DR: The proteolytic systems hijacked by SARS-CoV for S protein activation are reviewed, their contribution to viral spread in the host is discussed and antiviral strategies targeting these enzymes are outlined.
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Effects of tannins source (Vaccinium vitis idaea L.) on rumen microbial fermentation in vivo
TL;DR: Results suggest that Vaccinium vitis idaea, as a source of tannins, has the potential to reduce rumen CH4 production and ammonia concentration.
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DESC1 and MSPL Activate Influenza A Viruses and Emerging Coronaviruses for Host Cell Entry
Pawel Zmora,Paulina Blazejewska,Anna Sophie Moldenhauer,Kathrin Welsch,Inga Nehlmeier,Qingyu Wu,Heike Schneider,Stefan Pöhlmann,Stephanie Bertram +8 more
TL;DR: It is demonstrated that two cellular serine proteases, DESC1 and MSPL, activate influenza viruses and emerging coronaviruses in cell culture and, because of their expression in human lung tissue, might promote viral spread in the infected host.
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Camelina sativa cake improved unsaturated fatty acids in ewe's milk
Malgorzata Szumacher-Strabel,Adam Cieślak,Pawel Zmora,Emilia Pers-Kamczyc,Sylwia Bielińska,M. Stanisz,Jacek Wójtowski +6 more
TL;DR: Milk from CSC-supplemented ewes was characterized by increased levels of beneficial nutritional factors, including mono- and n-3 polyunsaturated fatty acids, and was also by lower atherogenic and thrombogenic indices.
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TMPRSS11A activates the influenza A virus hemagglutinin and the MERS coronavirus spike protein and is insensitive against blockade by HAI-1.
Pawel Zmora,Markus Hoffmann,Heike Kollmus,Anna-Sophie Moldenhauer,Olga Danov,Armin Braun,Michael Winkler,Klaus Schughart,Klaus Schughart,Stefan Pöhlmann,Stefan Pöhlmann +10 more
TL;DR: The results suggest that TMPRSS11A could promote FLUAV spread in target cells and that HA-activating TTSPs exhibit differential sensitivity to blockade by cellular serine protease inhibitors.