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Pieter A. Doevendans

Researcher at University of Michigan

Publications -  29
Citations -  1528

Pieter A. Doevendans is an academic researcher from University of Michigan. The author has contributed to research in topics: Myocardial infarction & Embryonic stem cell. The author has an hindex of 15, co-authored 29 publications receiving 1447 citations. Previous affiliations of Pieter A. Doevendans include Howard Hughes Medical Institute & Maastricht University Medical Centre.

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Quantification of epicardial and peri-coronary fat using cardiac computed tomography; reproducibility and relation with obesity and metabolic syndrome in patients suspected of coronary artery disease.

TL;DR: Volumetric quantification of EAT using cardiac CT is highly reproducible compared to more simple measurements as EAT and peri-coronary fat thickness and area, and the quantity of E AT and pero-corOnary fat is related with the presence of obesity and metabolic syndrome in patients suspected of CAD.

Expert consensus document from the European Society of Cardiology on catheter-based renal

TL;DR: This expert consensus document summarizes the view of an expert panel of the European Society of Cardiology and the European Association of Percutaneous Cardiovascular Interventions to provide guidance regarding guidance regarding treatment options for patients with resistant hypertension.
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CETP genotype predicts increased mortality in statin-treated men with proven cardiovascular disease: an adverse pharmacogenetic interaction.

TL;DR: In statin-treated male CAD patients, genetic variation conferring low CETP levels is associated with increased 10-year mortality, suggesting that efficacy of statin therapy to reduce cardiovascular risk depends on CETP genotype and associated CETP plasma levels.
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Electrocardiographic diagnosis of reperfusion during thrombolytic therapy in acute myocardial infarction

TL;DR: Electrocardiographic monitoring in patients receiving thrombolytic therapy for an acute myocardial infarction revealed early signs of reperfusion of the infarct-related vessel, and a increase in ST-segment deviation was recorded in 61% of the patients, whereas 69% had an increase in chest pain preceding the eventual decline.