P
Priscilla Kincaid-Smith
Researcher at Royal Melbourne Hospital
Publications - 160
Citations - 4160
Priscilla Kincaid-Smith is an academic researcher from Royal Melbourne Hospital. The author has contributed to research in topics: Pregnancy & Renal function. The author has an hindex of 34, co-authored 160 publications receiving 4090 citations. Previous affiliations of Priscilla Kincaid-Smith include Queen Victoria Hospital & University of Melbourne.
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Evaluation of ultrasonographic diagnostic criteria for autosomal dominant polycystic kidney disease 1
TL;DR: DNA linkage among subjects from 128 sibships within 18 PKD1 families was used as the basis for an assessment of ultrasound sensitivity, and the presence of at least two renal cysts in individuals at risk and younger than 30 years may be regarded as sufficient to establish a diagnosis.
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Phenotype and genotype heterogeneity in autosomal dominant polycystic kidney disease.
D Ravine,Rowan G. Walker,Robert N Gibson,SM Forrest,Robert I. Richards,K Friend,Leslie J. Sheffield,Priscilla Kincaid-Smith,D. M. Danks +8 more
TL;DR: Non-PKD1 ADPKD has a much milder phenotype than that linked to PKD1, and the reported prevalence of this genotype is probably an underestimate.
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The Clinical Course of Mesangial IgA Associated Nephropathy in Adults
TL;DR: The rate of clinical deterioration correlated with proteinuria, hypertension, impaired renal function, crescents and sclerosed glomeruli on biopsy, and continuing high urinary erythrocyte counts were the strongest predictor of a progressive course.
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Lupus Anticoagulant Associated with Renal Thrombotic Microangiopathy and Pregnancy-Related Renal Failure
TL;DR: The clinical features in 12 women with a circulating coagulation inhibitor (lupus 'anticoagulant') were studied and correlated with findings in 22 renal biopsies in these patients, and it is proposed that these lesions are characteristic of the renal findings in patients with a lupus '.
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Anticoagulants in "irreversible" acute renal failure.
TL;DR: 6 consecutive cases of oliguric renal failure shown histologically to be due to glomerulonephritis or obstructive lesions in arterioles and glomeruli improved considerably following continuous high-dose infusion of heparin, which was given in addition to steroids and immunosuppressive drugs.