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Priyank Shukla

Researcher at University of Florida

Publications -  26
Citations -  597

Priyank Shukla is an academic researcher from University of Florida. The author has contributed to research in topics: Basal ganglia & Putamen. The author has an hindex of 11, co-authored 18 publications receiving 432 citations. Previous affiliations of Priyank Shukla include The Mind Research Network & Medical University of South Carolina.

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Free-water imaging in Parkinson’s disease and atypical parkinsonism

TL;DR: A novel bi-tensor diffusion analysis model was used to indicate that all forms of parkinsonism had elevated free-water in the substantia nigra, and both multiple system atrophy and progressive supranuclear palsy, but not Parkinson's disease, showed a broad network of elevatedFree-water and altered free- water corrected fractional anisotropy that included the basal ganglia, thalamus, and cerebellum.
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Functional Brain Activity Relates to 0–3 and 3–8 Hz Force Oscillations in Essential Tremor

TL;DR: Significant differences in brain activity were observed between essential tremor and Parkinson's disease, and functional connectivity analyses during the task evidenced reduced cerebellar-cortical functional connectivity in patients withessential tremor compared with controls and Parkinson’s disease.
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Distinct Functional and Macrostructural Brain Changes in Parkinson’s Disease and Multiple System Atrophy

TL;DR: Results provide the first evidence that fMRI activation is abnormal in the basal ganglia, cerebellum, and cerebrum in MSAp, and that a key distinguishing feature between MSA p and PD is the extensive and widespread volume loss throughout the brain in M SAp.
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Functional activity of the sensorimotor cortex and cerebellum relates to cervical dystonia symptoms

TL;DR: Findings show widespread changes in functional brain activity in CD and most importantly that dystonic symptoms relate to disrupted activity in the somatosensory cortex and cerebellum.
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Distinct patterns of brain activity in progressive supranuclear palsy and Parkinson's disease.

TL;DR: It is found that PSP and PD share reduced functional activity of the basal ganglia and cortical motor areas, but this is more pronounced in PSP than in PD.