Q
Qing Jiang
Researcher at Purdue University
Publications - 46
Citations - 2157
Qing Jiang is an academic researcher from Purdue University. The author has contributed to research in topics: Vitamin E & Tocotrienol. The author has an hindex of 21, co-authored 41 publications receiving 1629 citations.
Papers
More filters
Journal ArticleDOI
Natural forms of vitamin E: metabolism, antioxidant, and anti-inflammatory activities and their role in disease prevention and therapy
TL;DR: This review focuses on non-αT forms of vitamin E with respect to their metabolism, anti-inflammatory effects and mechanisms, and in vivo efficacy in preclinical models as well as human clinical intervention studies.
Journal ArticleDOI
Long-chain carboxychromanols, metabolites of vitamin E, are potent inhibitors of cyclooxygenases
TL;DR: Long-chain carboxychromanols, including 13′-carboxy chromanol, are novel cyclooxygenase inhibitors, may serve as anti-inflammation and anticancer agents, and may contribute to the beneficial effects of certain forms of vitamin E.
Journal ArticleDOI
Gamma-tocotrienol induces apoptosis and autophagy in prostate cancer cells by increasing intracellular dihydrosphingosine and dihydroceramide
TL;DR: It was shown that γTE treatment promoted apoptosis, necrosis and autophagy in human prostate PC‐3 and LNCaP cancer cells and support the notion that elevation of intracellular diHydroceramide and dihydrosphingosine is likely a novel anticancer mechanism.
Journal ArticleDOI
Natural Forms of Vitamin E as Effective Agents for Cancer Prevention and Therapy
TL;DR: The existing evidence strongly indicates that these lesser-known vitamin E forms are effective agents for cancer prevention or as adjuvants for improving prevention, therapy, and control of cancer.
Journal ArticleDOI
Natural forms of vitamin E and 13'-carboxychromanol, a long-chain vitamin E metabolite, inhibit leukotriene generation from stimulated neutrophils by blocking calcium influx and suppressing 5-lipoxygenase activity, respectively.
TL;DR: It is found that δT prevented ionophore-caused cytoplasmic membrane disruption, which may account for its blocking of calcium influx, and these activities by vitamin E forms and long-chain carboxychromanol provide potential molecular bases for the differential anti-inflammatory effects ofitamin E forms in vivo.